🏆 Finalist — NIH Data Sharing Index (“S-Index”) Challenge

Madeline N. Hayes

University of Toronto

ORCID: 0000-0002-0089-8995

Also affiliated with Hospital for Sick Children

MedicineBiochemistry, Genetics and Molecular BiologyImmunology and Microbiology

Pilot corpus only

This score is computed over theSindex pilot corpus and does not cover the full scientific literature. Scores are relative to papers we have ingested — papers, authors, and institutions outside the pilot are not represented. Methodology.

Top 100%percentile
0Author DataRank

Indexed papers

0in pilot corpus
datarank_citation_only_1hop_v6· scope data_onlyMethodology

Papers

Driven by 4 papers. Top paper: “Single-cell analysis and functional characterization uncover the stem cell hierarchies and developmental origins of rhabdomyosarcoma”.

Single-cell analysis and functional characterization uncover the stem cell hierarchies and developmental origins of rhabdomyosarcoma

Nature Cancer(2022)10.1038/s43018-022-00414-w
N/A
0.631DataRank · unranked
66 citations

Qian Qin, Chuan Yan, Madeline N. Hayes, Sara P. Garcia, Haibin Xi +16 more

A syngeneic spontaneous zebrafish model of tp53-deficient, EGFRvIII, and PI3KCAH1047R-driven glioblastoma reveals inhibitory roles for inflammation during tumor initiation and relapse in vivo

eLife(2024)10.7554/elife.93077
N/A
0.498DataRank · unranked
11 citations

Cassandra D’Amata, Bret J. Pearson, Madeline N. Hayes, A. H. Weiss

Defining function of wild-type and three patient-specific TP53 mutations in a zebrafish model of embryonal rhabdomyosarcoma

eLife(2023)10.7554/elife.68221
N/A
0.358DataRank · unranked
7 citations

Kunal Baxi, Amanda Lipsitt, Nicole R. Hensch, Long Wang, Prethish Sreenivas +17 more

A syngeneic spontaneous zebrafish model of tp53-deficient, EGFRvIII, and PI3KCAH1047R-driven glioblastoma reveals inhibitory roles for inflammation during tumor initiation and relapse in vivo

eLife(2024)10.7554/elife.93077.3
N/A
0DataRank · unranked
0 citations

Cassandra D’Amata, Bret J. Pearson, Madeline N. Hayes, A. H. Weiss