Ben Cocanougher

Cincinnati Children's Hospital Medical Center

Also affiliated with Duke University, Duke Medical Center, University of Cincinnati Medical Center

Immunology and MicrobiologyNeuroscienceBiochemistry, Genetics and Molecular BiologyMedicineAgricultural and Biological Sciences

Pilot corpus only

This score is computed over theSindex pilot corpus and does not cover the full scientific literature. Scores are relative to papers we have ingested — papers, authors, and institutions outside the pilot are not represented. Methodology.

Top 100%percentile

0Author DataRank

Indexed papers

0in pilot corpus

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Papers

Driven by 5 papers. Top paper: “PHKA1-associated phosphorylase kinase deficiency: a monogenic disorder of exercise intolerance and myalgia”.

PHKA1-associated phosphorylase kinase deficiency: a monogenic disorder of exercise intolerance and myalgia

npj Genomic Medicine(2025)10.1038/s41525-025-00527-y

N/A

0.104DataRank · unranked

1 citations

Angie H Fares, Ben Cocanougher, Jamie K. Lim, Andrea B. Haijer‐Schreuder, Terry G. J. Derks +5 more

Single cell RNA-seq analysis reveals temporally-regulated and quiescence-regulated gene expression in Drosophila larval neuroblasts

Neural Development(2022)10.1186/s13064-022-00163-7

N/A

0DataRank · unranked

36 citations

Ben Cocanougher, Chhavi Sood, Xin Yuan, Andrea B. Kohn, Leonid L. Moroz +4 more

Single substitution in H3.3G34 alters DNMT3A recruitment to cause progressive neurodegeneration

Cell(2023)10.1016/j.cell.2023.02.023

N/A

0DataRank · unranked

68 citations

Carol Chen, Augusto Faria Andrade, Nisha Kabir, Pariya Azarafshar, Shahir M. Morcos +40 more

The severity of MUSK pathogenic variants is predicted by the protein domain they disrupt

Human Genetics and Genomics Advances(2024)10.1016/j.xhgg.2024.100288

N/A

0DataRank · unranked

3 citations

Samuel W Liu, Ludmila Francescatto, Alexander Behura, Mariele Anneling, David G. Jackson +8 more

The recurrent deep intronic pseudoexon-inducing variant COL6A1 c.930+189C>T results in a consistently severe phenotype of COL6-related dystrophy: Towards clinical trial readiness for splice-modulating therapy

medRxiv(2024)10.1101/2024.03.29.24304673

N/A

0DataRank · unranked

2 citations

Véronique Bolduc, Fady Guirguis, Sandra Donkervoort, Ying Hu, Rotem Orbach +64 more