Dextran Sulfate Sodium (DSS)‐Induced Colitis in Mice is a research paper published in Current Protocols in Immunology (2014). On theSindex it has a DataRank of 1.1. It has been cited 2,117 times.
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Base Score Contribution
1.1
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
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Grant: K01 DK083275
PHS HHS
Grant: R03 094864
NIDDK NIH HHS
Grant: R01 DK097865
MeSH Terms
Keywords
Additional file 2 of Facilitation of colonic T cell immune responses is associated with an exacerbation of dextran sodium sulfate–induced colitis in mice lacking microsomal prostaglandin E synthase-1
Additional file 2 of Facilitation of colonic T cell immune responses is associated with an exacerbation of dextran sodium sulfate–induced colitis in mice lacking microsomal prostaglandin E synthase-1
Additional file 1 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 1 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 2 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 2 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 3 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 3 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 4 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 4 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 5 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 5 of The role of T cell trafficking in CTLA-4 blockade-induced gut immunopathology
Additional file 1 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 1 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 2 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 2 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 3 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 3 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 4 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits
Additional file 4 of Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits