Proton extrusion during oxidative burst in microglia exacerbates pathological acidosis following traumatic brain injury is a research paper published in Glia (2020). On theSindex it has a DataRank of 0. It has been cited 94 times.
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National Institutes of Health
Grant: 2R01 NR013601
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Grant: F32 NS105355
National Institutes of Health
Grant: K99 NS116032
National Institutes of Health
Grant: R01 NS088627
National Institutes of Health
Grant: R01 NS094527
National Institutes of Health
Grant: R01 NS110567
National Institutes of Health
Grant: R01 NS110635
National Institutes of Health
Grant: R01 NS110825
National Institutes of Health
Grant: RF1 NS110637
NINDS NIH HHS
Grant: RF1 NS094527
NINR NIH HHS
Grant: R01 NR013601
National Institutes of Health
Grant: 5F32NS105355-02
Injury-induced microglia/macrophage senescence and chronic immune dysfunction in TBI
National Institutes of Health
Grant: 5R01NS110825-05
The Function and Mechanisms of Voltage-Gated Proton Channel Hv1 in Spinal Cord Injury
National Institutes of Health
Grant: 5R01NS110635-05
Mechanism of Inflammatory Related Brain Dysfunction after Spinal Cord Injury
National Institutes of Health
Grant: 5K99NS116032-02
Mechanisms of injury-induced senescence and immune-sequelae in chronic TBI
National Institutes of Health
Grant: 5R01NS088627-06
The Role of Microglia in Epilepsy
National Institutes of Health
Grant: 3R01NS094527-05S1
The new roles of the autophagy-lysosomal pathway in spinal cord injury-mediated dementia
National Institutes of Health
Grant: 1RF1NS110637-01
Dementia Following Spinal Cord Injury: Mechanism and Therapeutic Targeting
National Institutes of Health
Grant: 5R01NS110567-04
Targeting brain inflammation and neurocognitive dysfunction in sepsis
National Institutes of Health
Grant: 1R01NR013601-01
Spinal Mechanisms Underlying SCI-Induced Pain: Implications for Targeted Therapy
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Additional file 1 of Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury
Additional file 1 of Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury
Additional file 1 of Microglial–oligodendrocyte interactions in myelination and neurological function recovery after traumatic brain injury
Additional file 1 of Microglial–oligodendrocyte interactions in myelination and neurological function recovery after traumatic brain injury
Additional file 1 of Deletion of Slc9a1 in Cx3cr1+ cells stimulated microglial subcluster CREB1 signaling and microglia-oligodendrocyte crosstalk
Additional file 1 of Deletion of Slc9a1 in Cx3cr1+ cells stimulated microglial subcluster CREB1 signaling and microglia-oligodendrocyte crosstalk
Additional file 1 of Bi-directional neuro-immune dysfunction after chronic experimental brain injury
Additional file 1 of Bi-directional neuro-immune dysfunction after chronic experimental brain injury
Additional file 1 of Sexually dimorphic extracellular vesicle responses after chronic spinal cord injury are associated with neuroinflammation and neurodegeneration in the aged brain
Additional file 1 of Sexually dimorphic extracellular vesicle responses after chronic spinal cord injury are associated with neuroinflammation and neurodegeneration in the aged brain
Additional file 2 of Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury
Additional file 2 of Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury