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Cln1‐mutations suppress Rab7‐RILP interaction and impair autophagy contributing to neuropathology in a mouse model of infantile neuronal ceroid lipofuscinosis

Journal of Inherited Metabolic Disease(2020)10.1002/jimd.12242Source: DataRank Database

Cln1‐mutations suppress Rab7‐RILP interaction and impair autophagy contributing to neuropathology in a mouse model of infantile neuronal ceroid lipofuscinosis is a research paper published in Journal of Inherited Metabolic Disease (2020). On theSindex it has a DataRank of 0. It has been cited 21 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access21 citations

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Linked data & code

gen refseq igsr nct

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 1.81)OA: greenTopics (52)IDs (PubMed, PMC, OpenAlex)Annotations (4)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (8)

Tamal Sadhukhan,Maria B. Bagh ORCID,Abhilash P. Appu ORCID,Goutam Chandra ORCID,Avisek Mondal ORCID

Live enrichment

PubMed:32279353 PMC:PMC8261861 OpenAlex:https://openalex.org/W3016174944

Intramural NIH HHS

Grant: Z99 HD999999

National Institutes of Health

National Institute of Child Health and Human Development

FWCI

1.81

Citation Percentile

0.9%

Citation Trend

2021

2022

2023

2024

2025

2026

greenLicense: http://onlinelibrary.wiley.com/termsAndConditions#vor

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/8261861

publisherhttps://api.wiley.com/onlinelibrary/tdm/v1/articles/10.1002%2Fjimd.12242

publisherhttps://onlinelibrary.wiley.com/doi/pdf/10.1002/jimd.12242

publisherhttps://onlinelibrary.wiley.com/doi/full-xml/10.1002/jimd.12242

Fields of Study

Lysosomal Storage Disorders ResearchCellular transport and secretionCalcium signaling and nucleotide metabolismAdaptor Proteins, Signal TransducingAnimalsAutophagyCells, CulturedDisease Models, AnimalLysosomesMiceMice, Inbred C57BLMice, KnockoutMutationNeuronal Ceroid-LipofuscinosesThiolester Hydrolasesrab GTP-Binding Proteinsrab7 GTP-Binding Proteins

MeSH Terms

rab7 GTP-Binding ProteinsAnimalsAutophagyCells, CulturedDisease Models, AnimalLysosomesMice, Inbred C57BLMutationNeuronal Ceroid-LipofuscinosesThiolester HydrolasesMice, Knockoutrab GTP-Binding ProteinsAdaptor Proteins, Signal TransducingMice

Keywords

Neuronal ceroid lipofuscinosisAutophagyNeuropathologyBatten diseaseLafora diseaseMutationBiologyCell biologyNeuroscienceGeneticsMedicineApoptosisDiseasePathologyGenePhosphorylationNeurodegenerationInfantile neuronal ceroid lipofuscinosisLysosomal Storage DiseaseS-palmitoylationPalmitoyl-protein Thioesterases-1

Sustainable Development Goals

SDG 0: Good health and well-being