Frontline Science: The expression of integrin αDβ2 (CD11d/CD18) on neutrophils orchestrates the defense mechanism against endotoxemia and sepsis is a research paper published in Journal of Leukocyte Biology (2021). On theSindex it has a DataRank of 0.559. It has been cited 15 times, with 9 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Base Score Contribution
0.416
From this paper's citation signal
Citation Network Contribution
0.143
From 6 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 9 citers.
National Institutes of Health
Grant: C06RR0306551
NIGMS NIH HHS
Grant: R01 GM122934
NIH HHS
Grant: RO1GM083016
NIDDK NIH HHS
Grant: R01 DK102020
National Institutes of Health
Grant: RO1GM119197
NIH HHS
Grant: RO1GM122934
NIGMS NIH HHS
Grant: R01 GM119197
NIGMS NIH HHS
Grant: R01 GM083016
National Institutes of Health
Grant: 1R01DK102020-01
Role of beta2 integrins in macrophage retention and egress during inflammation
National Institutes of Health
Grant: 5R01GM119197-04
Training innate immunity: A new approach to the treatment of sepsis
National Institutes of Health
Grant: 3R01GM122934-04S1
Equipment Supplement Requisition for Mesoscale Quick Plex SQ 120mm Reader
Tennessee State University
FWCI
0.73
Citation Percentile
0.7%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Cholinergic signaling via the α7 nicotinic acetylcholine receptor regulates the migration of monocyte-derived macrophages during acute inflammation
Additional file 1 of Cholinergic signaling via the α7 nicotinic acetylcholine receptor regulates the migration of monocyte-derived macrophages during acute inflammation