Intracellular calcium leak as a therapeutic target for RYR1-related myopathies is a research paper published in Acta Neuropathologica (2020). On theSindex it has a DataRank of 0.635. It has been cited 68 times.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Base Score Contribution
0.635
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institutes of Health
Grant: T32HL120826
National Institutes of Health
Grant: UL1TR001873
National Institutes of Health
Grant: R01HL145473
National Institutes of Health
Grant: R01DK118240
National Institutes of Health
Grant: R01HL142903
National Institutes of Health
Grant: R01HL061503
National Institutes of Health
Grant: R01HL140934
National Institutes of Health
Grant: R01AR070194
National Institutes of Health
Grant: R25NS076445
National Institutes of Health
Grant: 5T32HL120826-10
Training in Cardiovascular Translational Research
National Institutes of Health
Grant: 5R01DK118240-03
Calcium and the physiology of diabetes
National Institutes of Health
Grant: 3UL1TR001873-07S1
Clinical and Translational Science Award
National Institutes of Health
Grant: 3R01HL145473-02S1
Structure-function analysis for elucidating pathogenicity of cardiac ryanodine receptor genetic variants
National Institutes of Health
Grant: 5R01HL061503-04
MOLECULAR BASIS OF HEART FAILURE
National Institutes of Health
Grant: 1R01HL142903-01A1
Ryanodine Receptor Defects in Cardiomyopathy Caused by Lamin A/C Gene Mutations
National Institutes of Health
Grant: 1R01HL140934-01
Exploring the Molecular Physiology of Atrial Fibrillation
National Institutes of Health
Grant: 1R25NS076445-01A1
Summer Program for Under Represented Students (SPURS)
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of RyR1-related myopathy mutations in ATP and calcium binding sites impair channel regulation
Additional file 1 of RyR1-related myopathy mutations in ATP and calcium binding sites impair channel regulation
Additional file 4 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 4 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 1 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 3 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 1 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 3 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 2 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion
Additional file 2 of Ryanodine receptor RyR1-mediated elevation of Ca2+ concentration is required for the late stage of myogenic differentiation and fusion