APOE4 exacerbates α-synuclein seeding activity and contributes to neurotoxicity in Alzheimer’s disease with Lewy body pathology is a research paper published in Acta Neuropathologica (2022). On theSindex it has a DataRank of 0.646. It has been cited 73 times.
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Base Score Contribution
0.646
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institute of Neurological Disorders and Stroke
Grant: U54NS110435
National Institute on Aging
Grant: U19AG069701
National Institutes of Health
Grant: 1U19AG069701-01
Biology and pathobiology of apoE in aging and Alzheimer's disease
National Institutes of Health
Grant: 5U54NS110435-02
Synergistic Interaction of amyloid-beta and alpha-synuclein in Lewy body Dementia
The Little Family Foundation
Mayo Alzheimer’s Disease Research Center Developmental Grant
Ted Turner and Family
The Mayo Clinic Center of Indiviudualized Medicine and an MSA Coalition award
FWCI
6.60
Citation Percentile
1.0%
Influential Citations
5
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of LRP1 is a neuronal receptor for α-synuclein uptake and spread
Additional file 1 of LRP1 is a neuronal receptor for α-synuclein uptake and spread
Additional file 1 of Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis
Additional file 1 of Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis
Additional file 1 of Association between APOE-ε4 allele and cognitive function is mediated by Alzheimer’s disease pathology: a population-based autopsy study in an admixed sample
Additional file 1 of Association between APOE-ε4 allele and cognitive function is mediated by Alzheimer’s disease pathology: a population-based autopsy study in an admixed sample