Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Not ‘Inactive’ After All: Cardiotoxic Mechanisms of Catecholamine Metabolism by Monoamine Oxidase

Cardiovascular Toxicology(2025)10.1007/s12012-025-10021-7Source: DataRank Database

Not ‘Inactive’ After All: Cardiotoxic Mechanisms of Catecholamine Metabolism by Monoamine Oxidase is a research paper published in Cardiovascular Toxicology (2025). On theSindex it has a DataRank of 0.330. It has been cited 8 times.

N/A

0.330DataRank · unranked

Scored on demand from live citation data

Open Access8 citations

Download PDF

Cite:

Linked data & code

doi

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)OA: hybridTopics (26)IDs (PubMed, PMC)Annotations (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

0.330

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.

Learn more about DataRank methodology →

Authors (2)

Ethan J. Anderson ORCID,Rachel M Crawford ORCID

Live enrichment

PubMed:40495031 PMC:PMC12267328

American Heart Association

Grant: 20SFRN35200003

National Heart, Lung, and Blood Institute

Grant: R01HL167087

National Institutes of Health

Grant: 5R01HL167087-02

Determinants of cardioprotection by circulating prohibitin-1 during sepsis

hybridLicense: cc-by

publisherhttps://link.springer.com/content/pdf/10.1007/s12012-025-10021-7.pdf

publisherhttps://link.springer.com/article/10.1007/s12012-025-10021-7/fulltext.html

repositoryhttps://pmc.ncbi.nlm.nih.gov/articles/PMC12267328/pdf/12012_2025_Article_10021.pdf

Europe_PMChttps://europepmc.org/articles/PMC12267328

Europe_PMChttps://europepmc.org/articles/PMC12267328?pdf=render

Fields of Study

0303 health sciences03 medical and health sciences

MeSH Terms

MyocardiumAnimalsHumansHeart DiseasesCatecholaminesMonoamine OxidaseMonoamine Oxidase InhibitorsSignal TransductionCardiotoxicity

Keywords

AmmoniaMitochondriaHydrogen peroxideMonoamine oxidaseCardiovascular diseaseCardiotoxicityCatecholaldehydeCatecholaminesMonoamine Oxidase InhibitorsHeart DiseasesMyocardiumHumansAnimalsReviewSignal Transduction

Not ‘Inactive’ After All: Cardiotoxic Mechanisms of Catecholamine Metabolism by Monoamine Oxidase

FAIR Checklist

DataRank Breakdown

Authors (2)

Funding (3 grants)

Open Access

Topics

Annotations (Europe PMC)