Endothelial Glycocalyx-Mediated Intercellular Interactions: Mechanisms and Implications for Atherosclerosis and Cancer Metastasis is a research paper published in Cardiovascular Engineering and Technology (2020). On theSindex it has a DataRank of 0. It has been cited 33 times.
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National Heart, Lung, and Blood Institute
Grant: K01 HL125499
Division of Civil, Mechanical and Manufacturing Innovation
Grant: CMMI-1846962
Division of Graduate Education
Grant: DGE-1451070
National Institutes of Health
Grant: 5K01HL125499-05
Atheroprotective vs. Atherogenic Glycocalyx Mechanotransduction Mechanisms
National Science Foundation
Grant: 1846962
CAREER: EMBRACE STEM (Endothelial MechanoBiology Research And multiCultural Education in STEM)
National Science Foundation
Grant: 1451070
Graduate Research Fellowship Program (GRFP)
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 1 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 2 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 2 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 3 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 3 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 4 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 4 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 5 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 6 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 7 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 6 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 7 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients
Additional file 5 of Communication between alveolar macrophages and fibroblasts via the TNFSF12-TNFRSF12A pathway promotes pulmonary fibrosis in severe COVID-19 patients