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Inhibiting thrombin improves motor function and decreases oxidative stress in the LRRK2 transgenic Drosophila melanogaster model of Parkinson’s disease

Biochemical and Biophysical Research Communications(2020)10.1016/j.bbrc.2020.04.068Source: DataRank Database

Inhibiting thrombin improves motor function and decreases oxidative stress in the LRRK2 transgenic Drosophila melanogaster model of Parkinson’s disease is a research paper published in Biochemical and Biophysical Research Communications (2020). On theSindex it has a DataRank of 0. It has been cited 23 times.

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0DataRank · unranked

Scored on demand from live citation data

23 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (4 grants)Impact (FWCI 1.09)OA: closedTopics (48)Linked Datasets (6)DataCite Reuse (6)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (0/2)

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

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Authors (4)

Jaclyn Iannucci ORCID,Navindra P. Seeram ORCID,Paula Grammas ORCID,Shelby L. Johnson ORCID

Live enrichment

PubMed:32423817 OpenAlex:https://openalex.org/W3024280076

NIA NIH HHS

Grant: R21 AG040718

National Institutes of Health

Grant: 1R21NS110628-01

National Institute of General Medical Sciences

Grant: P20GM103430

Salve Regina University

FWCI

1.09

Citation Percentile

0.8%

Citation Trend

2020

2021

2022

2023

2024

2025

2026

closedLicense: https://www.elsevier.com/legal/tdmrep-license

publisherhttps://api.elsevier.com/content/article/PII:S0006291X20307944?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0006291X20307944?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.bbrc.2020.04.068

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32423817

Fields of Study

Parkinson's Disease Mechanisms and TreatmentsMosquito-borne diseases and controlSignaling Pathways in DiseaseAnimalsAnimals, Genetically ModifiedAntithrombinsDabigatranDisease Models, AnimalDrosophila melanogasterFemaleHumansLeucine-Rich Repeat Serine-Threonine Protein Kinase-2LocomotionMaleMutationOxidative StressParkinson DiseaseThrombin

MeSH Terms

DabigatranLeucine-Rich Repeat Serine-Threonine Protein Kinase-2AnimalsAntithrombinsDisease Models, AnimalDrosophila melanogasterFemaleHumansLocomotionMaleMutationParkinson DiseaseThrombinOxidative StressAnimals, Genetically Modified

Keywords

Oxidative stressNeuroinflammationPharmacologyParkinson's diseaseDirect thrombin inhibitorNeuroprotectionMedicineNADPH oxidaseInternal medicineEndocrinologyBiologyDabigatranDiseaseAtrial fibrillationParkinson’s Disease

Sustainable Development Goals

SDG 0: Good health and well-being

Additional file 1 of NOX4-mediated astrocyte ferroptosis in Alzheimer’s disease

10.6084/m9.figshare.26741912.v1figshare

Additional file 1 of NOX4-mediated astrocyte ferroptosis in Alzheimer’s disease

10.6084/m9.figshare.26741912figshare

Additional file 2 of NOX4-mediated astrocyte ferroptosis in Alzheimer’s disease

10.6084/m9.figshare.26741915.v1figshare

Additional file 2 of NOX4-mediated astrocyte ferroptosis in Alzheimer’s disease

10.6084/m9.figshare.26741915figshare

Additional file 3 of NOX4-mediated astrocyte ferroptosis in Alzheimer’s disease

10.6084/m9.figshare.26741918.v1figshare