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Metabolic regulation of EGFR effector and feedback signaling in pancreatic cancer cells requires K-Ras

Biochemical and Biophysical Research Communications(2020)10.1016/j.bbrc.2020.09.029Source: DataRank Database

Metabolic regulation of EGFR effector and feedback signaling in pancreatic cancer cells requires K-Ras is a research paper published in Biochemical and Biophysical Research Communications (2020). On theSindex it has a DataRank of 0. It has been cited 5 times.

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Open Access5 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (8 grants)Impact (FWCI 0.35)OA: greenTopics (54)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (2)

Cosimo Commisso ORCID,Szu-Wei Lee ORCID

Live enrichment

PubMed:32972751 PMC:PMC7655618 OpenAlex:https://openalex.org/W3087618217

NCI NIH HHS

Grant: P30 CA030199

NCI NIH HHS

Grant: R01 CA207189

NCI NIH HHS

Grant: T32 CA211036

NCI NIH HHS

Grant: R21 CA243701

National Institutes of Health

Grant: 5R01CA207189-03

Regulation of Nutrient Stress-Induced Macropinocytosis in Pancreatic Ductal Adenocarcinoma

National Institutes of Health

Grant: 1R21CA243701-01A1

The Macropinosome: Uncovering the Molecular Anatomy of an Oncogene-driven Organelle

European Commission

Grant: 207189

Identification and analysis of novel proteins required for Wingless secretion.

National Cancer Institute

FWCI

0.35

Citation Percentile

0.5%

Citation Trend

2021

2022

greenLicense: Elsevier TDM

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7655618

journalhttps://doi.org/10.1016/j.bbrc.2020.09.029

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32972751

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7655618

Fields of Study

Cancer, Hypoxia, and MetabolismPancreatic and Hepatic Oncology ResearchMetabolism, Diabetes, and Cancer0301 basic medicine03 medical and health sciences

MeSH Terms

GlutamineHumansPancreatic NeoplasmsProto-Oncogene Proteins p21(ras)MAP Kinase Signaling SystemCarcinoma, Pancreatic DuctalFeedback, PhysiologicalCell Line, TumorExtracellular Signal-Regulated MAP KinasesProto-Oncogene Proteins c-aktErbB ReceptorsHeparin-binding EGF-like Growth Factor

Keywords

Cell biologyGlutamineSignal transductionBiologyMAPK/ERK pathwayPhosphorylationProtein kinase BEffectorContext (archaeology)Receptor tyrosine kinaseCyclin-dependent kinase 8KRASEpidermal growth factor receptorCancer researchReceptorCancerBiochemistryNotch signaling pathwayAmino acidGeneticsMetabolismEGFRRasERKAKTNutrient StressFeedback, PhysiologicalMAP Kinase Signaling SystemErbB ReceptorsPancreatic NeoplasmsProto-Oncogene Proteins p21(ras)Cell Line, TumorHumansExtracellular Signal-Regulated MAP KinasesProto-Oncogene Proteins c-aktCarcinoma, Pancreatic DuctalHeparin-binding EGF-like Growth Factor

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being