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Co-targeting of HDAC, PI3K, and Bcl-2 results in metabolic and transcriptional reprogramming and decreased mitochondrial function in acute myeloid leukemia

Biochemical Pharmacology(2022)10.1016/j.bcp.2022.115283Source: DataRank Database

Co-targeting of HDAC, PI3K, and Bcl-2 results in metabolic and transcriptional reprogramming and decreased mitochondrial function in acute myeloid leukemia is a research paper published in Biochemical Pharmacology (2022). On theSindex it has a DataRank of 0. It has been cited 22 times.

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Open Access22 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (12 grants)Impact (FWCI 2.81)OA: greenTopics (57)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (16)

Xinan Qiao,Xinyu Li ORCID,Yongwei Su ORCID,Jenna L. Carter ORCID,Jun Ma ORCID

Live enrichment

PubMed:36208684 PMC:PMC10411618 OpenAlex:https://openalex.org/W4302009295

NCI

Grant: CA009531

NCI

Grant: P30 CA022453

NCI NIH HHS

Grant: T32 CA009531

National Institutes of Health

Grant: 3P30CA022453-37S2

Cancer Center Support Grant

National Institutes of Health

Grant: 2T32CA009531-21

Training Program in the Biology of Cancer

National Institutes of Health

Barbara Ann Karmanos Cancer Institute

RE Children’s Foundation

Wayne State University

School of Medicine, Wayne State University

New Brunswick Children's Foundation

Jilin University

FWCI

2.81

Citation Percentile

0.9%

Citation Trend

2023

2024

2025

2026

greenLicense: Elsevier TDM

repositoryhttps://pmc.ncbi.nlm.nih.gov/articles/PMC10411618/pdf/nihms-1920965.pdf

publisherhttps://api.elsevier.com/content/article/PII:S000629522200377X?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S000629522200377X?httpAccept=text/plain

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/10411618

Fields of Study

Acute Myeloid Leukemia ResearchHistone Deacetylase Inhibitors ResearchCancer, Hypoxia, and Metabolism0301 basic medicine03 medical and health sciencesMiceAnimalsPhosphatidylinositol 3-KinasesCell Line, TumorLeukemia, Myeloid, AcuteCytarabineBridged Bicyclo Compounds, HeterocyclicMitochondriaApoptosisSulfonamides

MeSH Terms

AnimalsCytarabineMitochondriaSulfonamidesLeukemia, Myeloid, AcuteApoptosisBridged Bicyclo Compounds, HeterocyclicPhosphatidylinositol 3-KinasesCell Line, TumorMice

Keywords

Myeloid leukemiaCancer researchPI3K/AKT/mTOR pathwayBiologyReprogrammingMyeloidLeukemiaTranscriptomeClofarabineVenetoclaxOxidative phosphorylationApoptosisPharmacologyImmunologyCytarabineCellGeneBiochemistryGene expressionChronic lymphocytic leukemiac-mycAcute myeloid leukemiaCudc-907Cytarabine (Arac) ResistanceMicePhosphatidylinositol 3-KinasesLeukemia, Myeloid, AcuteSulfonamidesCell Line, TumorAnimalsBridged Bicyclo Compounds, HeterocyclicMitochondria

Sustainable Development Goals

SDG 3: 3. Good health

Co-targeting of HDAC, PI3K, and Bcl-2 results in metabolic and transcriptional reprogramming and decreased mitochondrial function in acute myeloid leukemia

FAIR Checklist

Authors (16)

Funding (12 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)