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Hypoxia: Friend or Foe for drug delivery in Pancreatic Cancer

Cancer Letters(2020)10.1016/j.canlet.2020.07.041Source: DataRank Database

Hypoxia: Friend or Foe for drug delivery in Pancreatic Cancer is a research paper published in Cancer Letters (2020). On theSindex it has a DataRank of 0. It has been cited 97 times.

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Open Access97 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (14 grants)Impact (FWCI 5.09)OA: greenTopics (52)Linked Datasets (25)DataCite Reuse (25)IDs (PubMed, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

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Reusable (0/3)

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Authors (4)

Brett C. Sheppard ORCID,Rosalie C. Sears ORCID,Adam W. G. Alani ORCID,Vidhi M. Shah ORCID

Live enrichment

PubMed:32822815 OpenAlex:https://openalex.org/W3052273069

National Institutes of Health

Grant: 5U2CCA233280-05

Omic and Multidimensional Spatial Atlas of Metastatic Breast and Prostate Cancers

National Institutes of Health

Grant: 5U01CA224012-04

Comparative analysis between patient-derived models of pancreatic ductal adenocarcinomas and matched tumor specimens

National Institutes of Health

Grant: 1R37CA234006-01A1

Novel Nanomedicine-Based Therapeutic Approach For Treatment of Cancer Cachexia

National Institutes of Health

Grant: 5R01CA186241-09

DeSUMOylation regulation of c-Myc

National Institutes of Health

Grant: 1R15CA227754-01A1

Mitigation of Doxorubicin-Induced Cardiotoxicity by Phytochemicals in Micelles

National Institutes of Health

Grant: 5R01CA196228-03

The Role of post-translational activation of Myc in pancreatic cancer

National Institutes of Health

Grant: 3U54CA209988-03S1

Measuring, Modeling and Controlling Heterogeneity

NCI NIH HHS

Grant: U01 CA224012

NCI NIH HHS

Grant: U2C CA233280

NCI NIH HHS

Grant: R15 CA227754

NCI NIH HHS

Grant: U54 CA209988

NCI NIH HHS

Grant: R01 CA196228

NCI NIH HHS

Grant: R37 CA234006

NCI NIH HHS

Grant: R01 CA186241

FWCI

5.09

Citation Percentile

1.0%

Citation Trend

2020

2021

2022

2023

2024

2025

2026

greenLicense: Elsevier TDM

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7879337

publisherhttps://api.elsevier.com/content/article/PII:S0304383520303979?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0304383520303979?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.canlet.2020.07.041

Fields of Study

Cancer, Hypoxia, and MetabolismPancreatic and Hepatic Oncology ResearchCancer Research and Treatments0301 basic medicine0303 health sciences03 medical and health sciencesCarcinoma, Pancreatic DuctalCell HypoxiaDrug Resistance, NeoplasmExtracellular MatrixHumansHypoxia-Inducible Factor 1, alpha SubunitPancreatic NeoplasmsTumor Microenvironment

MeSH Terms

Extracellular MatrixHumansPancreatic NeoplasmsCell HypoxiaDrug Resistance, NeoplasmCarcinoma, Pancreatic DuctalHypoxia-Inducible Factor 1, alpha SubunitTumor Microenvironment

Keywords

DesmoplasiaPancreatic cancerHypoxia (environmental)Cancer researchMalignancyTumor microenvironmentStromaTumor hypoxiaHypoxia-inducible factorsPancreatic tumorBiologyTumor progressionDrug deliveryMedicineCancerPathologyInternal medicineImmunologyRadiation therapyTumor cellsChemistryImmunohistochemistryGenePancreatic NeoplasmsDrug Resistance, NeoplasmHumansHypoxia-Inducible Factor 1, alpha SubunitCell HypoxiaCarcinoma, Pancreatic DuctalExtracellular Matrix

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

Additional file 1 of RETSAT associates with DDX39B to promote fork restarting and resistance to gemcitabine based chemotherapy in pancreatic ductal adenocarcinoma

10.6084/m9.figshare.21129972.v1figshare

Additional file 1 of RETSAT associates with DDX39B to promote fork restarting and resistance to gemcitabine based chemotherapy in pancreatic ductal adenocarcinoma

10.6084/m9.figshare.21129972figshare

Additional file 9 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644360figshare

Additional file 9 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644360.v1figshare

Additional file 8 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644357figshare

Additional file 7 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644354.v1figshare

Additional file 8 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644357.v1figshare

Additional file 6 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644351figshare

Additional file 6 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644351.v1figshare

Additional file 5 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644348.v1figshare

Additional file 3 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644342figshare

Additional file 5 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644348figshare

Additional file 7 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644354figshare

Additional file 3 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644342.v1figshare

Additional file 4 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644345.v1figshare

Additional file 4 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.26644345figshare

Additional file 1 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.24630276.v1figshare

Additional file 1 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.24630276figshare

Additional file 2 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.24630430figshare

Additional file 2 of Genomic analysis reveals HDAC1 regulates clinically relevant transcriptional programs in Pancreatic cancer

10.6084/m9.figshare.24630430.v1figshare