Functional Long Non-coding RNAs Evolve from Junk Transcripts is a research paper published in Cell (2020). On theSindex it has a DataRank of 0. It has been cited 271 times.
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Natural Sciences and Engineering Research Council of Canada
Grant: unidentified
unidentified
National Institutes of Health
Natural Sciences and Engineering Research Council of Canada
U.S. National Library of Medicine
Canadian Institutes of Health Research
CIHR
FWCI
14.14
Citation Percentile
1.0%
Influential Citations
5
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 8 of Global analysis of expression, maturation and subcellular localization of mouse liver transcriptome identifies novel sex-biased and TCPOBOP-responsive long non-coding RNAs
Additional file 8 of Global analysis of expression, maturation and subcellular localization of mouse liver transcriptome identifies novel sex-biased and TCPOBOP-responsive long non-coding RNAs
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Additional file 1 of Analysis of differentially expressed long non-coding RNAs in LPS-induced human HMC3 microglial cells
Additional file 1 of Analysis of differentially expressed long non-coding RNAs in LPS-induced human HMC3 microglial cells
Additional file 2 of Analysis of differentially expressed long non-coding RNAs in LPS-induced human HMC3 microglial cells
Additional file 2 of Analysis of differentially expressed long non-coding RNAs in LPS-induced human HMC3 microglial cells
Additional file 1 of Blockade of the lncRNA-DOT1L-LAMP5 axis enhances autophagy and promotes degradation of MLL fusion proteins
Additional file 1 of Blockade of the lncRNA-DOT1L-LAMP5 axis enhances autophagy and promotes degradation of MLL fusion proteins
Additional file 1 of ADAMTS9-AS1 inhibits tumor growth and drug resistance in clear cell renal cell carcinoma via recruiting HuR to enhance ADAMTS9 mRNA stability
Additional file 1 of ADAMTS9-AS1 inhibits tumor growth and drug resistance in clear cell renal cell carcinoma via recruiting HuR to enhance ADAMTS9 mRNA stability
Additional file 5 of m6A modified BACE1-AS contributes to liver metastasis and stemness-like properties in colorectal cancer through TUFT1 dependent activation of Wnt signaling
Additional file 1 of m6A modified BACE1-AS contributes to liver metastasis and stemness-like properties in colorectal cancer through TUFT1 dependent activation of Wnt signaling
Additional file 3 of m6A modified BACE1-AS contributes to liver metastasis and stemness-like properties in colorectal cancer through TUFT1 dependent activation of Wnt signaling
Additional file 5 of m6A modified BACE1-AS contributes to liver metastasis and stemness-like properties in colorectal cancer through TUFT1 dependent activation of Wnt signaling