Genome-wide functional screen of 3′UTR variants uncovers causal variants for human disease and evolution is a research paper published in Cell (2021). On theSindex it has a DataRank of 0. It has been cited 188 times.
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NHGRI
Grant: F32HG00922
NHGRI
Grant: K99HG010669
NHGRI
Grant: R00HG008179
NHGRI
Grant: UM1HG009435
NHGRI
Grant: DEB-1401237
NHGRI
Grant: F30GM114940
NHGRI
Grant: R37AI147868
NHGRI NIH HHS
Grant: R00 HG010669
NHGRI NIH HHS
Grant: F32 HG009226
National Institutes of Health
Grant: 5K99HG010669-02
Comprehensive Characterization of Adaptive Regulatory Variation Linked to Human Disease
National Science Foundation
Grant: 1401237
DISSERTATION RESEARCH: Validation of positive selection on gene regulation at the genome-wide scale
National Institutes of Health
Grant: 5F30GM114940-02
Genome-wide validation of posttranscriptional variation in selection and disease
National Institutes of Health
Grant: 1UM1HG009435-01
Comprehensive functional characterization and dissection of noncoding regulatory elements and human genetic variation
National Institutes of Health
Grant: 4R00HG008179-03
Massively Parallel Phenotypic Characterization of Non-coding Genetic Variation
National Institutes of Health
Grant: 5R37AI147868-05
The HUSH complex in HIV-1 latency
Howard Hughes Medical Institute
Howard Hughes Medical Institute
FWCI
10.75
Citation Percentile
1.0%
Citation Trend
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Sustainable Development Goals
Additional file 5 of New insights into genome annotation in Podospora anserina through re-exploiting multiple RNA-seq data
Additional file 5 of New insights into genome annotation in Podospora anserina through re-exploiting multiple RNA-seq data
Additional file 2 of New insights into genome annotation in Podospora anserina through re-exploiting multiple RNA-seq data
Additional file 2 of New insights into genome annotation in Podospora anserina through re-exploiting multiple RNA-seq data
Additional file 1 of Functional genomic analysis delineates regulatory mechanisms of GWAS-identified bipolar disorder risk variants
Additional file 1 of Functional genomic analysis delineates regulatory mechanisms of GWAS-identified bipolar disorder risk variants
Additional file 8 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 6 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 6 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 7 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 8 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 9 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 7 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 9 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 4 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 2 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 5 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 4 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 5 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier
Additional file 2 of Pumilio-1 mediated translational control of claudin-5 at the blood-brain barrier