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FOXK1 Participates in DNA Damage Response by Controlling 53BP1 Function

Cell Reports(2020)10.1016/j.celrep.2020.108018Source: DataRank Database

FOXK1 Participates in DNA Damage Response by Controlling 53BP1 Function is a research paper published in Cell Reports (2020). On theSindex it has a DataRank of 0. It has been cited 20 times.

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Scored on demand from live citation data

Open Access20 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (14 grants)Impact (FWCI 1.04)OA: goldTopics (66)IDs (PubMed, PMC, OpenAlex)Annotations (4)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (11)

Xu Feng ORCID,Guangsheng Pei ORCID,Mrinal Srivastava ORCID,Chao Wang ORCID,Zhen Chen ORCID

Live enrichment

PubMed:32783940 PMC:PMC7458625 OpenAlex:https://openalex.org/W3048939192

University of Texas Health Science Center at Houston

Grant: RP180734

Cancer Prevention and Research Institute of Texas

Grant: RP160667

Cancer Prevention and Research Institute of Texas

Grant: P30CA016672

Cancer Prevention and Research Institute of Texas

Grant: P01CA193124

Cancer Prevention and Research Institute of Texas

Grant: R01CA210929

Cancer Prevention and Research Institute of Texas

Grant: R01CA216437

National Institutes of Health

Grant: R01CA216911

National Institutes of Health

Grant: 3P30CA016672-41S4

Cancer Center Support (CORE) Grant

National Institutes of Health

Grant: 5R01CA216911-03

Novel regulations of DNA damage repair

National Institutes of Health

Grant: 5R01CA210929-03

Define redundant functions of H2AX and NBS1 in DNA repair

National Institutes of Health

Grant: 5R01CA216437-03

53BP1-dependent pathway in DNA repair

National Institutes of Health

Grant: 5P01CA193124-06

Processing and Repair of DNA Crosslinks

Cancer Prevention and Research Institute of Texas

National Institutes of Health

FWCI

1.04

Citation Percentile

0.8%

Citation Trend

2021

2022

2023

2024

2025

goldLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.celrep.2020.108018

publisherhttps://doi.org/10.1016/j.celrep.2020.108018

publisherhttps://api.elsevier.com/content/article/PII:S2211124720310032?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2211124720310032?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32783940

Fields of Study

DNA Repair MechanismsPARP inhibition in cancer therapyFOXO transcription factor regulation0301 basic medicine0303 health sciences03 medical and health sciencesAtaxia Telangiectasia Mutated ProteinsBRCA1 ProteinDNA Breaks, Double-StrandedDNA End-Joining RepairDNA RepairForkhead Transcription FactorsGene Knockout TechniquesHEK293 CellsHeLa CellsHomologous RecombinationHumansPhosphorylationTelomeric Repeat Binding Protein 2Tumor Suppressor p53-Binding Protein 1

MeSH Terms

Tumor Suppressor p53-Binding Protein 1DNA RepairHeLa CellsHumansPhosphorylationBRCA1 ProteinTelomeric Repeat Binding Protein 2Forkhead Transcription FactorsDNA Breaks, Double-StrandedGene Knockout TechniquesHEK293 CellsHomologous RecombinationDNA End-Joining RepairAtaxia Telangiectasia Mutated ProteinsHela Cells

Keywords

DNA damageHomologous recombinationPoly ADP ribose polymeraseDNA repairCell biologyDNAChemistryPolymeraseTelomereShelterinMolecular biologyBiologyDNA-binding proteinGeneTranscription factorBiochemistryDNA End-Joining RepairQH301-705.5BRCA1 ProteinForkhead Transcription FactorsAtaxia Telangiectasia Mutated ProteinsArticleGene Knockout TechniquesHEK293 CellsHumansDNA Breaks, Double-StrandedTelomeric Repeat Binding Protein 2Biology (General)PhosphorylationTumor Suppressor p53-Binding Protein 1HeLa Cells

Sustainable Development Goals

SDG 16: 16. Peace & justiceSDG 0: Good health and well-being