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The mitochondrial UPR regulator ATF5 promotes intestinal barrier function via control of the satiety response

Cell Reports(2022)10.1016/j.celrep.2022.111789Source: DataRank Database

The mitochondrial UPR regulator ATF5 promotes intestinal barrier function via control of the satiety response is a research paper published in Cell Reports (2022). On theSindex it has a DataRank of 0. It has been cited 20 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access20 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 1.29)OA: goldTopics (50)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (6)

Siraje Arif Mahmud ORCID,Aundrea K. Westfall ORCID,Todd A. Castoe ORCID,Rance E. Berg ORCID,Mark W. Pellegrino ORCID

Live enrichment

PubMed:36516750 PMC:PMC9805788 OpenAlex:https://openalex.org/W4311310359

National Institutes of Health

Grant: R35GM128885

Cancer Prevention and Research Institute of Texas

Grant: RR160053

National Institutes of Health

Grant: 5R35GM128885-05

Mechanisms of host protection during infection via the mitochondrial unfolded protein response

FWCI

1.29

Citation Percentile

0.8%

Citation Trend

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1016/j.celrep.2022.111789

publisherhttps://doi.org/10.1016/j.celrep.2022.111789

publisherhttps://api.elsevier.com/content/article/PII:S2211124722016771?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2211124722016771?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36516750

Fields of Study

Genetics, Aging, and Longevity in Model OrganismsCircadian rhythm and melatoninEndoplasmic Reticulum Stress and Disease0301 basic medicine0303 health sciences03 medical and health sciencesAnimalsCaenorhabditis elegans ProteinsSatiety ResponseCaenorhabditis elegansMitochondriaUnfolded Protein ResponseMammals

MeSH Terms

AnimalsMammalsMitochondriaSatiety ResponseCaenorhabditis elegansCaenorhabditis elegans ProteinsUnfolded Protein Response

Keywords

Unfolded protein responseBiologyCaenorhabditis elegansCell biologyBarrier functionMitochondrionTranscription factorRegulatorSecretionEndoplasmic reticulumEndocrinologyBiochemistryGeneMitochondriaCholecystokininLeptinColitishyperglycemiaSatietyHost-pathogen InteractionEpithelial BarrierAtf5Upr(mt)Cp: Molecular BiologyCp: MetabolismMammalsAnimalsCaenorhabditis elegans ProteinsSatiety ResponseArticle

Sustainable Development Goals

SDG 0: Good health and well-being

The mitochondrial UPR regulator ATF5 promotes intestinal barrier function via control of the satiety response

FAIR Checklist

Authors (6)

Funding (3 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)