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Enhanced bypass of PD-L1 translation reduces the therapeutic response to mTOR kinase inhibitors

Cell Reports(2023)10.1016/j.celrep.2023.112764Source: DataRank Database

Enhanced bypass of PD-L1 translation reduces the therapeutic response to mTOR kinase inhibitors is a research paper published in Cell Reports (2023). On theSindex it has a DataRank of 0. It has been cited 16 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access16 citations

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Linked data & code

gen cellosaurus refseq uniprot

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (15 grants)Impact (FWCI 1.50)OA: goldTopics (44)IDs (PubMed, PMC, OpenAlex)Annotations (4)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (4)

Qing Ye ORCID,Murong Ma,Qing‐Bai She ORCID,Yanan Cao ORCID

Live enrichment

PubMed:37405918 PMC:PMC10491412 OpenAlex:https://openalex.org/W4383101367

NIH

Grant: T32CA165990

NIH

Grant: R01CA175105

NIH

Grant: R21ES031712

NIH

Grant: P20M121327

NIH

Grant: R01CA203257

NIGMS NIH HHS

Grant: P20 GM121327

NCI NIH HHS

Grant: P30 CA177558

NCATS NIH HHS

Grant: UL1 TR001998

National Institutes of Health

Grant: 1R21ES031712-01

Translational Control in Cr(VI) Carcinogenesis

National Institutes of Health

Grant: 5R01CA175105-08

Targeting Translation Dependence in Colorectal Cancer Progression

National Institutes of Health

Grant: 3T32CA165990-02S2

Interdisciplinary Research Training in Cancer Biology

National Institutes of Health

Grant: 3P30CA177558-01S1

CTRP Supplement

National Institutes of Health

Grant: 1P20GM121327-01

University of Kentucky Center for Cancer and Metabolism

National Institutes of Health

Grant: 5R01CA203257-04

Natural product-based modulators of 4E-BP1 phosphorylation

Markey Cancer Center, University of Kentucky

FWCI

1.50

Citation Percentile

0.8%

Citation Trend

2024

2025

2026

goldLicense: cc-by-nc-nd

journalhttp://www.cell.com/article/S2211124723007751/pdf

publisherhttp://www.cell.com/article/S2211124723007751/pdf

publisherhttps://api.elsevier.com/content/article/PII:S2211124723007751?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2211124723007751?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.celrep.2023.112764

Fields of Study

PI3K/AKT/mTOR signaling in cancerChronic Lymphocytic Leukemia ResearchMelanoma and MAPK Pathways03 medical and health sciences0302 clinical medicine

MeSH Terms

Mechanistic Target of Rapamycin Complex 1MTOR InhibitorsHumansNeoplasmsCell Line, TumorTOR Serine-Threonine KinasesB7-H1 Antigen

Keywords

PI3K/AKT/mTOR pathwayTranslation (biology)KinaseChemistryProtein kinase ACancer researchCell biologyPharmacologyMedicineSignal transductionBiologyBiochemistryGeneMessenger RNAIRESPD-L1immunosuppressionMtor4e-bp1Eif4aCp: Molecular BiologyCp: CancerQH301-705.5TOR Serine-Threonine KinasesMTOR InhibitorsMechanistic Target of Rapamycin Complex 1ArticleB7-H1 AntigenNeoplasmsCell Line, TumorHumansBiology (General)

Sustainable Development Goals

SDG 0: Good health and well-being