Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Base-excision repair pathway regulates transcription-replication conflicts in pancreatic ductal adenocarcinoma

Cell Reports(2024)10.1016/j.celrep.2024.114820Source: DataRank Database

Base-excision repair pathway regulates transcription-replication conflicts in pancreatic ductal adenocarcinoma is a research paper published in Cell Reports (2024). On theSindex it has a DataRank of 0. It has been cited 7 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access7 citations

Free full text

Cite:

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (7 grants)Impact (FWCI 1.26)OA: goldTopics (52)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (12)

Tiane Li ORCID,Anup K. Singh ORCID,Yingying Wang ORCID,Marc A. Attiyeh ORCID,Fatemeh Kohram ORCID

Live enrichment

PubMed:39368091 OpenAlex:https://openalex.org/W4403115516

National Cancer Institute Division of Cancer Epidemiology and Genetics

Grant: R01 CA073764

Pancreatic Cancer Action Network

Grant: 20-20-RAOO

American Cancer Society

Grant: 132966-IRG

National Comprehensive Cancer Network Foundation

Grant: P062683

National Institutes of Health

Grant: P30CA033572

National Cancer Institute

National Comprehensive Cancer Network

FWCI

1.26

Citation Percentile

0.8%

Citation Trend

2025

2026

goldLicense: cc-by-sa

journalhttps://doi.org/10.1016/j.celrep.2024.114820

publisherhttps://doi.org/10.1016/j.celrep.2024.114820

publisherhttps://api.elsevier.com/content/article/PII:S2211124724011719?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2211124724011719?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39368091

Fields of Study

Pancreatic and Hepatic Oncology ResearchEpigenetics and DNA MethylationCancer-related Molecular PathwaysHumansCarcinoma, Pancreatic DuctalDNA RepairPancreatic NeoplasmsDNA ReplicationCell Line, TumorTranscription, GeneticDNA DamageAtaxia Telangiectasia Mutated ProteinsCell ProliferationRNA Polymerase IICheckpoint Kinase 1Signal TransductionExcision Repair

MeSH Terms

Checkpoint Kinase 1Excision RepairDNA DamageDNA RepairDNA ReplicationHumansPancreatic NeoplasmsRNA Polymerase IITranscription, GeneticSignal TransductionCarcinoma, Pancreatic DuctalCell Line, TumorCell ProliferationAtaxia Telangiectasia Mutated Proteins

Keywords

Pancreatic ductal adenocarcinomaTranscription (linguistics)Transcription factorBiologyCancer researchReplication (statistics)Cell biologyPancreatic cancerGeneticsGeneCancerVirologyDNA repairBase Excision RepairR-loopsReplication StressAtr PathwayOncogene-induced Replication StressCp: Molecular BiologyCp: CancerTranscription Replication Conflict

Sustainable Development Goals

SDG 0: Zero hunger