TRIM Proteins Regulate Autophagy and Can Target Autophagic Substrates by Direct Recognition is a research paper published in Developmental Cell (2014). On theSindex it has a DataRank of 9.7. It has been cited 334 times, with 200 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Base Score Contribution
0.872
From this paper's citation signal
Citation Network Contribution
8.9
From 200 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 200 citers.
National Institutes of Health
Grant: 5R01AI111935-07
Autophagy Against Tuberculosis and HIV
National Institutes of Health
Grant: 5P51OD011132-53
Support of Yerkes National Primate Research Center
National Institutes of Health
Grant: 3P51OD011132-64S2
Renovation to expand and enhance NHP housing for HIV/AIDS research at the Emory NPRC
National Institutes of Health
Grant: 5T32AI007538-13
Biology of Infectious Disease and Inflammation
National Institutes of Health
Grant: 5UL1TR000041-05
University of New Mexico Clinical and Translational Science Center
National Institutes of Health
Grant: 2R56CA109618-06
Beclin 1-Bcl-2 Interactions: Effects on Autophagy
NCI NIH HHS
Grant: CA109618
NCATS NIH HHS
Grant: UL1 TR000041
NIAID NIH HHS
Grant: R37 AI042999
NIAID NIH HHS
Grant: R01 AI111935
NIAID NIH HHS
Grant: AI111935
NIAID NIH HHS
Grant: T32 AI007538
NCI NIH HHS
Grant: R56 CA109618
NIAID NIH HHS
Grant: R01 AI042999
NIH HHS
Grant: P51 OD011132
NIAID NIH HHS
Grant: AI042999
NCI NIH HHS
Grant: R01 CA109618
FWCI
17.49
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma
Additional file 1 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma
Additional file 3 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma
Additional file 3 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma
Additional file 2 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 2 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 3 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 3 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 4 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 4 of GNIP1 functions both as a scaffold protein and an E3 ubiquitin ligase to regulate autophagy in lung cancer
Additional file 1 of Mechanism of autophagy induced by activation of the AMPK/ERK/mTOR signaling pathway after TRIM22-mediated DENV-2 infection of HUVECs
Additional file 1 of Mechanism of autophagy induced by activation of the AMPK/ERK/mTOR signaling pathway after TRIM22-mediated DENV-2 infection of HUVECs
Additional file 2 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma
Additional file 2 of Aloin induced apoptosis by enhancing autophagic flux through the PI3K/AKT axis in osteosarcoma