Glycolysis-Independent Glucose Metabolism Distinguishes TE from ICM Fate during Mammalian Embryogenesis is a research paper published in Developmental Cell (2020). On theSindex it has a DataRank of 0. It has been cited 187 times.
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NCI NIH HHS
Grant: R01 CA217608
NIH HHS
Grant: DP1 OD008356
NIDDK NIH HHS
Grant: DP1 DK098059
National Institutes of Health
Grant: 8DP1DK098059-02
Developmental Control of Metabolism
National Institutes of Health
Grant: 5R01CA217608-29
Metabolic and signaling control of tumorformation in drosophila
National Institute of Diabetes and Digestive and Kidney Diseases
Eli and Edythe Broad Center of Regenerative Medicine and Stem Cell Research, University of California Los Angeles
March of Dimes Foundation
FWCI
20.56
Citation Percentile
1.0%
Influential Citations
9
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos
Additional file 1 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos
Additional file 3 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos
Additional file 3 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos
Additional file 1 of Antioxidant procyanidin B2 protects oocytes against cryoinjuries via mitochondria regulated cortical tension
Additional file 1 of Antioxidant procyanidin B2 protects oocytes against cryoinjuries via mitochondria regulated cortical tension
Additional file 1 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 1 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 5 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 2 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 2 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 5 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 4 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 3 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 3 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 4 of mTOR hypoactivity leads to trophectoderm cell failure by enhancing lysosomal activation and disrupting the cytoskeleton in preimplantation embryo
Additional file 2 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos
Additional file 2 of A DNA methylation state transition model reveals the programmed epigenetic heterogeneity in human pre-implantation embryos