High Amount of Transcription Factor IRF8 Engages AP1-IRF Composite Elements in Enhancers to Direct Type 1 Conventional Dendritic Cell Identity is a research paper published in Immunity (2020). On theSindex it has a DataRank of 0. It has been cited 79 times.
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NCI NIH HHS
Grant: R01 CA248919
NCATS NIH HHS
Grant: UL1 TR000448
NIAID NIH HHS
Grant: R01 AI150297
NCATS NIH HHS
Grant: UL1 TR002345
NIAID NIH HHS
Grant: R01 AI162643
NCI NIH HHS
Grant: F31 CA228240
NCI NIH HHS
Grant: P30 CA091842
National Cancer Institute
National Institutes of Health
Howard Hughes Medical Institute
National Science Foundation
Howard Hughes Medical Institute
FWCI
3.15
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma
Additional file 1 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma
Additional file 3 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma
Additional file 3 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma
Additional file 2 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma
Additional file 2 of IRF8 deficiency-induced myeloid-derived suppressor cell promote immune evasion in lung adenocarcinoma