The complement system drives local inflammatory tissue priming by metabolic reprogramming of synovial fibroblasts is a research paper published in Immunity (2021). On theSindex it has a DataRank of 0. It has been cited 243 times.
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Deutsche Forschungsgemeinschaft
Grant: FG 2886\u2013TP04
Deutsche Forschungsgemeinschaft
Grant: CRC1181-B03
Deutsche Forschungsgemeinschaft
Grant: TI-3
Deutsche Forschungsgemeinschaft
Grant: EXC2167 PMI
European Research Council
Grant: 810316 4DnanoSCOPE
National Institute for Health Research (NIHR)
Grant: ACF-2011-09-001
Kennedy Trust
Grant: KENN192006
Academy of Medical Sciences
Grant: SGL021\1029
Kennedy Trust
Grant: KENN161704
Versus Arthritis
Grant: 22253
Medical Research Council
Grant: MR/S025308/1
Versus Arthritis
Grant: 22658
National Institute for Health Research (NIHR)
Grant: CL-2018-09-001
Deutsche Forschungsgemeinschaft
Grant: unidentified
unidentified
European Commission
Grant: 777357
Rheuma Tolerance for Cure
European Commission
Grant: 810316
Advancing osteoporosis medicine by observing bone microstructure and remodelling using a four-dimensional nanoscope
China Scholarship Council
European Commission
Pfizer
Roche
European Research Council
UCB
European Federation of Pharmaceutical Industries and Associations
National Institutes of Health
GlaxoSmithKline
FWCI
11.79
Citation Percentile
1.0%
Influential Citations
7
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Single-cell multimodal analysis identifies common regulatory programs in synovial fibroblasts of rheumatoid arthritis patients and modeled TNF-driven arthritis
Additional file 1 of Single-cell multimodal analysis identifies common regulatory programs in synovial fibroblasts of rheumatoid arthritis patients and modeled TNF-driven arthritis
Additional file 2 of Complement factor H attenuates TNF-α-induced inflammation by upregulating EIF3C in rheumatoid arthritis
Additional file 2 of Complement factor H attenuates TNF-α-induced inflammation by upregulating EIF3C in rheumatoid arthritis
Additional file 1 of Epigenetic targets of Janus kinase inhibitors are linked to genetic risks of rheumatoid arthritis
Additional file 1 of Epigenetic targets of Janus kinase inhibitors are linked to genetic risks of rheumatoid arthritis
Additional file 1 of Complement factor H attenuates TNF-α-induced inflammation by upregulating EIF3C in rheumatoid arthritis
Additional file 1 of Complement factor H attenuates TNF-α-induced inflammation by upregulating EIF3C in rheumatoid arthritis