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Transcription factor RORα enforces stability of the Th17 cell effector program by binding to a Rorc cis-regulatory element

Immunity(2022)10.1016/j.immuni.2022.09.013Source: DataRank Database

Transcription factor RORα enforces stability of the Th17 cell effector program by binding to a Rorc cis-regulatory element is a research paper published in Immunity (2022). On theSindex it has a DataRank of 0.677. It has been cited 90 times.

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0.677DataRank · unranked

Scored on demand from live citation data

Open Access90 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (5 grants)Impact (FWCI 5.73)OA: hybridTopics (26)Linked Datasets (10)DataCite Reuse (10)IDs (PubMed, PMC, OpenAlex)Annotations (1)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

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DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

0.677

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.

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Authors (8)

Maria Pokrovskii ORCID,Lina Kroehling ORCID,Boram Kim ORCID,Seung Yong Kim,Lin Wu ORCID

Live enrichment

PubMed:36243007 PMC:PMC9757081 OpenAlex:https://openalex.org/W4306179862

NIAID NIH HHS

Grant: R01 AI121436

NIDDK NIH HHS

Grant: R01 DK103358

NCI NIH HHS

Grant: T32 CA009161

NCI NIH HHS

Grant: P30 CA016087

Howard Hughes Medical Institute

FWCI

5.73

Citation Percentile

1.0%

Citation Trend

2023

2024

2025

2026

hybridLicense: cc-by

journalhttp://www.cell.com/article/S1074761322005076/pdf

publisherhttp://www.cell.com/article/S1074761322005076/pdf

publisherhttps://api.elsevier.com/content/article/PII:S1074761322005076?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S1074761322005076?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.immuni.2022.09.013

Fields of Study

IL-33, ST2, and ILC PathwaysImmune Cell Function and InteractionT-cell and B-cell Immunology

MeSH Terms

Cell DifferentiationEncephalomyelitis, Autoimmune, ExperimentalGene Expression RegulationTranscription FactorsNuclear Receptor Subfamily 1, Group F, Member 3Th17 Cells

Keywords

RAR-related orphan receptor gammaTranscription factorBiologyEffectorCell biologyCellular differentiationLineage (genetic)Orphan receptorGeneticsGeneHeat-labile enterotoxinGene regulationAutoimmunityexperimental autoimmune encephalomyelitisEAESegmented Filamentous BacteriaSfb

Additional file 1 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26094351.v1figshare

Additional file 1 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26094351figshare

Additional file 2 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26094469.v1figshare

Additional file 2 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26094469figshare

Additional file 3 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26738112.v1figshare

Additional file 3 of Dietary ellagic acid therapy for CNS autoimmunity: Targeting on Alloprevotella rava and propionate metabolism

10.6084/m9.figshare.26738112figshare