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NOX4 regulates macrophage apoptosis resistance to induce fibrotic progression

Journal of Biological Chemistry(2021)10.1016/j.jbc.2021.100810Source: DataRank Database

NOX4 regulates macrophage apoptosis resistance to induce fibrotic progression is a research paper published in Journal of Biological Chemistry (2021). On theSindex it has a DataRank of 0. It has been cited 21 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access21 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (12 grants)Impact (FWCI 1.96)OA: goldTopics (76)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (5)

Linlin Gu,Jungsoon Kang,A. Dhyani,A. Brent Carter ORCID,Jennifer L. Larson‐Casey ORCID

Live enrichment

PubMed:34023385 PMC:PMC8214193 OpenAlex:https://openalex.org/W3162814154

National Institutes of Health

Grant: 1 I01 CX001715-2

National Institutes of Health

Grant: 2R01 ES015981-13

National Institutes of Health

Grant: P01 HL114470-7

National Institutes of Health

Grant: P42 ES027723-2

NIEHS NIH HHS

Grant: P42 ES027723

NIEHS NIH HHS

Grant: R01 ES015981

CSRD VA

Grant: I01 CX001715

NIAMS NIH HHS

Grant: P30 AR048311

National Institutes of Health

Grant: 5R01ES015981-02

Lung Inflammation and Fibrosis After Asbestosis Exposure is Regulated by Rac1

National Institutes of Health

Grant: 5P30AR048311-08

Analytic and Preparative Cytometry Facility

National Institutes of Health

Grant: 5U01AI027667-14

HIV DISEASE RESEARCH AGENDA

National Institutes of Health

Grant: 1P42ES027723-01A1

Impact of Airborne Heavy Metals on Lung Disease and the Environment

FWCI

1.96

Citation Percentile

0.9%

Citation Trend

2021

2022

2023

2024

2025

2026

goldLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.jbc.2021.100810

publisherhttps://doi.org/10.1016/j.jbc.2021.100810

publisherhttps://api.elsevier.com/content/article/PII:S0021925821006050?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0021925821006050?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34023385

Fields of Study

Interstitial Lung Diseases and Idiopathic Pulmonary FibrosisOccupational and environmental lung diseasesOccupational exposure and asthma0301 basic medicine0303 health sciences03 medical and health sciencesAnimalsApoptosisCell LineDisease ProgressionFemaleIdiopathic Pulmonary FibrosisLungMacrophagesMaleMice, Inbred C57BLMitochondriaModels, BiologicalMonocytesNADPH Oxidase 4PhosphorylationReactive Oxygen Speciesbcl-Associated Death ProteinMice

MeSH Terms

NADPH Oxidase 4AnimalsCell LineFemaleLungMacrophagesMaleMice, Inbred C57BLMitochondriaModels, BiologicalMonocytesPhosphorylationApoptosisReactive Oxygen SpeciesDisease Progressionbcl-Associated Death ProteinMiceIdiopathic Pulmonary Fibrosis

Keywords

NOX4ApoptosisFibrosisPulmonary fibrosisMacrophageCancer researchReactive oxygen speciesBiologyPathogenesisNADPH oxidaseImmunologyCell biologyMedicinePathologyBiochemistryApoptosis ResistanceMonocyte-derived MacrophagesMaleMacrophagesModels, BiologicalIdiopathic Pulmonary FibrosisMonocytesCell LineMitochondriaMice, Inbred C57BLMiceNADPH Oxidase 4Disease ProgressionAnimalsFemalebcl-Associated Death ProteinPhosphorylationLungResearch Article

Sustainable Development Goals

SDG 3: 3. Good health