The innate immune kinase TBK1 directly increases mTORC2 activity and downstream signaling to Akt is a research paper published in Journal of Biological Chemistry (2021). On theSindex it has a DataRank of 1.0. It has been cited 28 times, with 27 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
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Base Score Contribution
0.505
From this paper's citation signal
Citation Network Contribution
0.498
From 21 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 27 citers.
National Institutes of Health
Grant: R01-DK-103877
National Institutes of Health
Grant: R01-GM-137577
American Diabetes Association
Grant: 1-12-BS-49
NIDDK NIH HHS
Grant: R56 DK126328
NIGMS NIH HHS
Grant: R01 GM137577
NIGMS NIH HHS
Grant: T32 GM145304
NIDDK NIH HHS
Grant: P30 DK020572
NIDDK NIH HHS
Grant: R01 DK103877
National Institutes of Health
Grant: 1R01DK103877-01
Direct regulation of mTORC1 and mTORC2 by the IKK-related kinases TBK1 and IKKe
National Institutes of Health
Grant: 5P30DK020572-37
ADMINISTRATIVE CORE
National Institutes of Health
Grant: 5R01GM137577-04
Unexpected role for AMPK and mTORC1 in cellular adaptation to nutrient stress
Michigan Diabetes Research Center, University of Michigan
FWCI
1.40
Citation Percentile
0.8%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of MARCH1 negatively regulates TBK1-mTOR signaling pathway by ubiquitinating TBK1
Additional file 1 of MARCH1 negatively regulates TBK1-mTOR signaling pathway by ubiquitinating TBK1