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Short-term CaMKII inhibition with tatCN19o does not erase pre-formed memory in mice and is neuroprotective in pigs

Journal of Biological Chemistry(2023)10.1016/j.jbc.2023.104693Source: DataRank Database

Short-term CaMKII inhibition with tatCN19o does not erase pre-formed memory in mice and is neuroprotective in pigs is a research paper published in Journal of Biological Chemistry (2023). On theSindex it has a DataRank of 0. It has been cited 9 times.

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Scored on demand from live citation data

Open Access9 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (19 grants)Impact (FWCI 1.00)OA: goldTopics (39)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (13)

Carolyn Nicole Brown ORCID,Tara B. Hendry‐Hofer ORCID,Thomas Rossetti,James E. Orfila ORCID,Jonathan E. Tullis

Live enrichment

PubMed:37037305 PMC:PMC10189404 OpenAlex:https://openalex.org/W4362721019

NIGMS

Grant: T32 GM007635

NINDS

Grant: F31 NS129254

NINDS

Grant: R01 NS081248

NINDS

Grant: R01 NS110383

NINDS

Grant: R01 NS118786

NIA

Grant: F31 AG069458

NIA

Grant: F32 AG066536

NIA

Grant: R01 AG067713

NIA

Grant: T32 AG000279

National Institutes of Health

Grant: 5F32AG066536-02

A-beta/APP signaling impairs CaMKII-dependent synaptic plasticity after ischemic brain injury

National Institutes of Health

Grant: 2T32AG000279-06A1

University of Colorado Aging Training Grant

National Institutes of Health

Grant: 5F31AG069458-02

CaMKII hypo-nitrosylatlon In age-related decline of synaptic plasticity and cognition

National Institutes of Health

Grant: 5R01NS081248-11

CaMKII Holoenzyme Mechanisms in Opposing Forms of Synaptic Plasticity

National Institutes of Health

Grant: 5T32GM007635-20

PREDOCTORAL TRAINING PROGRAM IN PHARMACOLOGY

National Institutes of Health

Grant: 5F31NS129254-02

Molecular computation by the CaMKII holoenzyme

National Institutes of Health

Grant: 5R01NS118786-03

CaMKII in global cerebral ischemia: mechanisms and therapeutic intervention

National Institutes of Health

Grant: 5R01AG067713-03

CaMKII nitrosylation in the age-related decline of synaptic plasticity

National Institutes of Health

Grant: 5R01NS110383-07

Postsynaptic kinase/phosphatase networks in amyloid beta-induced synaptic dysfunction

National Institutes of Health

FWCI

1.00

Citation Percentile

0.7%

Citation Trend

2024

2025

2026

goldLicense: cc-by

journalhttp://www.jbc.org/article/S0021925823003356/pdf

publisherhttp://www.jbc.org/article/S0021925823003356/pdf

publisherhttps://api.elsevier.com/content/article/PII:S0021925823003356?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0021925823003356?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.jbc.2023.104693

Fields of Study

Neuroscience and Neuropharmacology ResearchNicotinic Acetylcholine Receptors StudyIon channel regulation and function03 medical and health sciences0302 clinical medicine

MeSH Terms

AnimalsHippocampusMemoryNeuronsPeptidesPhosphorylationSwineMiceCalcium-Calmodulin-Dependent Protein Kinase Type 2

Keywords

NeuroprotectionNeuroscienceHippocampal formationVentricular fibrillationMedicinePharmacologyDefibrillationInternal medicineBiologyMemoryLearningMousePigContextual Fear ConditioningGlobal Cerebral IschemiaCa(2+)/calmodulin-dependent Protein Kinase Ii (Camkii)NeuronsMiceSwineAnimalsPhosphorylationCalcium-Calmodulin-Dependent Protein Kinase Type 2PeptidesHippocampusResearch Article

Sustainable Development Goals

SDG 0: Good health and well-being

Short-term CaMKII inhibition with tatCN19o does not erase pre-formed memory in mice and is neuroprotective in pigs

FAIR Checklist

Authors (13)

Funding (19 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)