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The histone methyltransferase KMT2D maintains cellular glucocorticoid responsiveness by shielding the glucocorticoid receptor from degradation

Journal of Biological Chemistry(2024)10.1016/j.jbc.2024.107581Source: DataRank Database

The histone methyltransferase KMT2D maintains cellular glucocorticoid responsiveness by shielding the glucocorticoid receptor from degradation is a research paper published in Journal of Biological Chemistry (2024). On theSindex it has a DataRank of 0. It has been cited 3 times.

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0DataRank · unranked

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Open Access3 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 0.48)OA: goldTopics (52)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (3)

Ferenc Livák ORCID,Jonathan D. Ashwell ORCID,Chuan-Jin Wu ORCID

Live enrichment

PubMed:39025450 PMC:PMC11350265 OpenAlex:https://openalex.org/W4400803724

National Cancer Institute

National Institutes of Health

Center for Cancer Research

FWCI

0.48

Citation Percentile

0.6%

Citation Trend

2025

goldLicense: cc-by

journalhttps://doi.org/10.1016/j.jbc.2024.107581

GOLDhttps://doi.org/10.1016/j.jbc.2024.107581

publisherhttps://doi.org/10.1016/j.jbc.2024.107581

publisherhttps://api.elsevier.com/content/article/PII:S0021925824020829?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0021925824020829?httpAccept=text/plain

Fields of Study

Cancer-related gene regulationProtein Degradation and InhibitorsCRISPR and Genetic EngineeringMedicineBiologyHumansReceptors, GlucocorticoidNeoplasm ProteinsGlucocorticoidsDNA-Binding ProteinsJurkat CellsProteolysisHistone-Lysine N-MethyltransferaseApoptosisCRISPR-Cas SystemsCell Line, Tumor

MeSH Terms

DNA-Binding ProteinsGlucocorticoidsHumansNeoplasm ProteinsHistone-Lysine N-MethyltransferaseReceptors, GlucocorticoidApoptosisJurkat CellsCell Line, TumorProteolysisCRISPR-Cas Systems

Keywords

BiologyJurkat cellsH3K4me3Histone methyltransferaseEpigeneticsChromatin immunoprecipitationHistone H3Glucocorticoid receptorHistoneCancer researchMethyltransferaseGene expressionMolecular biologyGeneCell biologyGeneticsMethylationPromoterT cellApoptosisLymphocytesGlucocorticoidsglucocorticoid resistanceProtein StabilityKmt2d

Sustainable Development Goals

SDG 0: Good health and well-being

The histone methyltransferase KMT2D maintains cellular glucocorticoid responsiveness by shielding the glucocorticoid receptor from degradation

FAIR Checklist

Authors (3)

Funding (3 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)