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A GluN2B disease-associated variant promotes the degradation of NMDA receptors via autophagy

Journal of Biological Chemistry(2026)10.1016/j.jbc.2026.111147Source: DataRank Database

A GluN2B disease-associated variant promotes the degradation of NMDA receptors via autophagy is a research paper published in Journal of Biological Chemistry (2026). On theSindex it has a DataRank of 0.

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Scored on demand from live citation data

Open Access0 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (5 grants)Impact (FWCI 16.42)OA: goldTopics (32)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (6)

Marnie P. Williams ORCID,Pei-Pei Zhang ORCID,Adrian J. Palumbo,Ting-Wei Mu ORCID,Adrian Palumbo

Live enrichment

PubMed:41525846 PMC:PMC12882719 OpenAlex:https://openalex.org/W7119791470

National Institute of Neurological Disorders and Stroke

Grant: F31NS139574

National Institute of Neurological Disorders and Stroke

Grant: R01NS117176

National Institute of Neurological Disorders and Stroke

Grant: R01NS105789

National Institute of Neurological Disorders and Stroke

Grant: R21NS145426

National Institutes of Health

FWCI

16.42

Citation Percentile

1.0%

Citation Trend

2026

goldLicense: cc-by

journalhttps://doi.org/10.1016/j.jbc.2026.111147

publisherhttps://doi.org/10.1016/j.jbc.2026.111147

publisherhttps://api.elsevier.com/content/article/PII:S0021925826000177?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0021925826000177?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/41525846

Fields of Study

Autophagy in Disease and TherapyNeuroscience and Neuropharmacology ResearchAutoimmune Neurological Disorders and TreatmentsReceptors, N-Methyl-D-AspartateAutophagyHumansProteolysisEndoplasmic ReticulumAnimalsHEK293 Cells

MeSH Terms

AnimalsAutophagyEndoplasmic ReticulumHumansLysosomesReceptors, N-Methyl-D-AspartateMutation, MissenseHEK293 CellsProteolysis

Keywords

ProteostasisAutophagyEndoplasmic reticulumProteolysisReceptorNMDA receptorNeurodegenerationMutagenesisProtein subunitNMDAREr-phagyLc3-interacting Region (Lir)Ccpg1

Sustainable Development Goals

SDG 0: Good health and well-being