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Chronic nSMase inhibition suppresses neuronal exosome spreading and sex-specifically attenuates amyloid pathology in APP knock-in Alzheimer's disease mice

Neurobiology of Disease(2023)10.1016/j.nbd.2023.106213Source: DataRank Database

Chronic nSMase inhibition suppresses neuronal exosome spreading and sex-specifically attenuates amyloid pathology in APP knock-in Alzheimer's disease mice is a research paper published in Neurobiology of Disease (2023). On theSindex it has a DataRank of 0. It has been cited 23 times.

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Scored on demand from live citation data

Open Access23 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (11 grants)Impact (FWCI 2.21)OA: goldTopics (59)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (11)

Francisco Espejo‐Porras ORCID,Shijie Jin ORCID,Zainuddin Quadri ORCID,Limin Wu ORCID,Marcela Bertolio ORCID

Live enrichment

PubMed:37364689 PMC:PMC10777534 OpenAlex:https://openalex.org/W4381950615

National Institutes of Health

Grant: AG059610

National Institutes of Health

Grant: AG078728

National Institutes of Health

Grant: NS125490

NINDS NIH HHS

Grant: R01 NS125490

NIA NIH HHS

Grant: RF1 AG057882

NIA NIH HHS

Grant: R01 AG078728

NIA NIH HHS

Grant: P30 AG072946

NIA NIH HHS

Grant: RF1 AG059610

National Institutes of Health

Grant: 5R01NS125490-02

Dysfunctional Astroglial Exosome to (motor) Neuron Axon Signaling in ALS

National Institutes of Health

Grant: 1RF1AG059610-01

Neuronal/Glial exosome signaling and beta-amyloid propagation in Alzheimer's Disease (AD)

National Institutes of Health

Grant: 5R01AG078728-04

CNS Exosomes-Mediated Adaptive Immunity in Alzheimer's Disease (AD)

FWCI

2.21

Citation Percentile

0.9%

Citation Trend

2023

2024

2025

2026

goldLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.nbd.2023.106213

GOLDhttps://doi.org/10.1016/j.nbd.2023.106213

publisherhttps://doi.org/10.1016/j.nbd.2023.106213

publisherhttps://api.elsevier.com/content/article/PII:S0969996123002280?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0969996123002280?httpAccept=text/plain

Fields of Study

Extracellular vesicles in diseaseSphingolipid Metabolism and SignalingMicroRNA in disease regulationMedicineBiology03 medical and health sciences0302 clinical medicine

MeSH Terms

Alzheimer DiseaseAnimalsDisease Models, AnimalFemaleMaleMice, TransgenicNeuronsAmyloid beta-PeptidesAmyloid beta-Protein PrecursorMiceExosomesPlaque, AmyloidAmyloidogenic Proteins

Keywords

CeramideExosomeHippocampusSphingolipidPresenilinEndocrinologyCognitive declineSphingomyelinAlzheimer's diseaseInternal medicineAmyloid (mycology)Amyloid precursor proteinBiologyMedicinePathologyDementiaDiseaseCell biologyMicrovesiclesBiochemistrymicroRNASexNsmaseMaleNeuronsAmyloid beta-PeptidesNeurosciences. Biological psychiatry. NeuropsychiatryMice, TransgenicPlaque, AmyloidAmyloidogenic ProteinsExosomesArticleMiceAmyloid beta-Protein PrecursorDisease Models, AnimalAlzheimer DiseaseAnimalsFemaleRC321-571

Sustainable Development Goals

SDG 0: Good health and well-being