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Formation of templated inclusions in a forebrain α-synuclein mouse model is independent of LRRK2

Neurobiology of Disease(2023)10.1016/j.nbd.2023.106338Source: DataRank Database

Formation of templated inclusions in a forebrain α-synuclein mouse model is independent of LRRK2 is a research paper published in Neurobiology of Disease (2023). On theSindex it has a DataRank of 0. It has been cited 5 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access5 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (1 grants)Impact (FWCI 0.78)OA: goldTopics (44)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (6)

Yue Ma ORCID,An Phu Tran Nguyen,Alina V. Offerman,Ian Beddows ORCID,Darren J. Moore ORCID

Live enrichment

PubMed:38435455 PMC:PMC10906965 OpenAlex:https://openalex.org/W4388006712

NIA NIH HHS

Grant: R56 AG074473

FWCI

0.78

Citation Percentile

0.7%

Citation Trend

2024

2025

goldLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.nbd.2023.106338

publisherhttps://doi.org/10.1016/j.nbd.2023.106338

publisherhttps://api.elsevier.com/content/article/PII:S0969996123003546?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0969996123003546?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/38435455

Fields of Study

Parkinson's Disease Mechanisms and TreatmentsNeurological diseases and metabolismBotulinum Toxin and Related Neurological DisordersAnimalsMicealpha-SynucleinDisease Models, AnimalMice, KnockoutParkinson DiseaseProsencephalonSynucleinopathiesLeucine-Rich Repeat Serine-Threonine Protein Kinase-2

MeSH Terms

Leucine-Rich Repeat Serine-Threonine Protein Kinase-2SynucleinopathiesAnimalsDisease Models, AnimalParkinson DiseaseProsencephalonMice, KnockoutMicealpha-Synuclein

Keywords

LRRK2NeurodegenerationParkinson's diseaseAlpha-synucleinNeuroscienceLewy bodyForebrainHippocampusPathologyNeuroinflammationDementia with Lewy bodiesKnockout mouseBiologyDiseaseMedicineDementiaCentral nervous systemGeneticsReceptorProtein aggregationMicrogliaAnimal ModelsParkinson’s Disease