Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Loss of mouse Stmn2 function causes motor neuropathy

Neuron(2022)10.1016/j.neuron.2022.02.011Source: DataRank Database

Loss of mouse Stmn2 function causes motor neuropathy is a research paper published in Neuron (2022). On theSindex it has a DataRank of 0. It has been cited 84 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access84 citations

Download PDF

Cite:

Linked data & code

gen

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (4 grants)Impact (FWCI 8.57)OA: bronzeTopics (60)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (19)

Leslie A. Nash ORCID,Kevin S. Smith ORCID,Marcel F. Leyton-Jaimes ORCID,Menglu Qian,Joseph R. Klim ORCID

Live enrichment

PubMed:35294901 PMC:PMC9119928 OpenAlex:https://openalex.org/W4220953393

NIA NIH HHS

Grant: K99 AG057808

NINDS NIH HHS

Grant: R01 NS089742

National Institutes of Health

Grant: 5K99AG057808-02

Exploring Mechanisms of Pathogenicity in C9ORF72 Frontotemporal Dementia and Amyotrophic Lateral Sclerosis

National Institutes of Health

Grant: 5R01NS089742-05

C9ORF72 in Motor System Biology and ALS

FWCI

8.57

Citation Percentile

1.0%

Citation Trend

2021

2022

2023

2024

2025

2026

bronzeLicense: Elsevier Non-Commercial

journalhttp://www.cell.com/article/S0896627322001763/pdf

publisherhttp://www.cell.com/article/S0896627322001763/pdf

publisherhttps://api.elsevier.com/content/article/PII:S0896627322001763?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0896627322001763?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.neuron.2022.02.011

Fields of Study

Amyotrophic Lateral Sclerosis ResearchRNA Research and SplicingNeurogenetic and Muscular Disorders Research03 medical and health sciences0302 clinical medicineAmyotrophic Lateral SclerosisAnimalsDisease Models, AnimalHomozygoteMiceMice, TransgenicMotor NeuronsNeuromuscular JunctionStathmin

MeSH Terms

Amyotrophic Lateral SclerosisAnimalsDisease Models, AnimalHomozygoteMice, TransgenicMotor NeuronsNeuromuscular JunctionStathminMice

Keywords

Amyotrophic lateral sclerosisLoss functionNeuromuscular junctionBiologyDenervationSpinal muscular atrophyPhenotypeMuscle atrophyMotor neuronCell biologyMutantNeuroscienceGeneRNA-binding proteinMotor systemRNAAtrophySpinal cordGeneticsAnatomyInternal medicineMedicineMicrotubulesALSCrisprMotor NeuropathyTardbpTdp43Scg10Stathmin 2Motor NeuronsHomozygoteMice, TransgenicDisease Models, AnimalMiceAnimalsStathmin

Sustainable Development Goals

SDG 0: Good health and well-being