Mitochondria dysfunction and metabolic reprogramming as drivers of idiopathic pulmonary fibrosis is a research paper published in Redox Biology (2020). On theSindex it has a DataRank of 0. It has been cited 269 times.
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National Institutes of Health
Grant: U01 HL145550-01
National Institutes of Health
Grant: R01 HL131789-03
National Institutes of Health
Grant: 5 R01 HL123766-04
National Institutes of Health
Grant: R01 HL149825
NHLBI NIH HHS
Grant: U01 HL145550
NHLBI NIH HHS
Grant: R01 HL123766
NHLBI NIH HHS
Grant: R01 HL131789
National Institutes of Health
Grant: 7U01HL145550-03
Mapping Age-Related Changes in the Lung
National Institutes of Health
Grant: 1R01HL149825-01
Type II Alveolar Redox Control in Fibrogenesis and Resolution
National Institutes of Health
Grant: 1R01HL123766-01A1
Aging of Mesenchymal Stem Cells Missing Link in IPF
National Institutes of Health
Grant: 5R01HL131789-04
Signaling mechanisms by which mitochondria regulates fibrosis in the lung
FWCI
11.26
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Lung immune signatures define two groups of end-stage IPF patients
Additional file 1 of Lung immune signatures define two groups of end-stage IPF patients
Additional file 1 of Modeling of lung phenotype of Hermansky–Pudlak syndrome type I using patient-specific iPSCs
Additional file 1 of Modeling of lung phenotype of Hermansky–Pudlak syndrome type I using patient-specific iPSCs
Additional file 1 of PRDX1 negatively regulates bleomycin-induced pulmonary fibrosis via inhibiting the epithelial-mesenchymal transition and lung fibroblast proliferation in vitro and in vivo
Additional file 1 of PRDX1 negatively regulates bleomycin-induced pulmonary fibrosis via inhibiting the epithelial-mesenchymal transition and lung fibroblast proliferation in vitro and in vivo
Additional file 5 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 5 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 6 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 6 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 2 of Lung immune signatures define two groups of end-stage IPF patients
Additional file 2 of Lung immune signatures define two groups of end-stage IPF patients
Additional file 2 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 3 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 3 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 4 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 4 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 2 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 1 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts
Additional file 1 of Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts