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Decreased autophagosome biogenesis, reduced NRF2, and enhanced ferroptotic cell death are underlying molecular mechanisms of non-alcoholic fatty liver disease

Redox Biology(2022)10.1016/j.redox.2022.102570Source: DataRank Database

Decreased autophagosome biogenesis, reduced NRF2, and enhanced ferroptotic cell death are underlying molecular mechanisms of non-alcoholic fatty liver disease is a research paper published in Redox Biology (2022). On theSindex it has a DataRank of 0. It has been cited 73 times.

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Open Access73 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (8 grants)Impact (FWCI 8.74)OA: goldTopics (79)Linked Datasets (2)DataCite Reuse (2)IDs (PubMed, PMC, OpenAlex)SDGs (3)

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Findable (2/2)

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Accessible (1/2)

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Interoperable (2/2)

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Authors (10)

Annadurai Anandhan ORCID,Jinjing Chen ORCID,Aryatara Shakya ORCID,Matthew Dodson ORCID,Aikseng Ooi ORCID

Live enrichment

PubMed:36495698 PMC:PMC9731892 OpenAlex:https://openalex.org/W4311765022

National Institutes of Health

Grant: P42ES004940

National Institutes of Health

Grant: P30 ES006694

National Institutes of Health

Grant: R35ES031575

National Institutes of Health

Grant: R01ES031463

National Institutes of Health

Grant: 1R01ES031463-01A1

Redox and Protein Homeostasis in Arsenic Tumorigenicity

National Institutes of Health

Grant: 3P42ES004940-14S4

HAZARDOUS WASTE RISK AND REMEDIATION IN THE SOUTHWEST

National Institutes of Health

Grant: 5P30ES006694-23

Southwest Environmental Health Sciences Center

National Institutes of Health

Grant: 5R35ES031575-04

NRF Transcription Factors in Environmental Stress and Disease Intervention

FWCI

8.74

Citation Percentile

1.0%

Citation Trend

2023

2024

2025

2026

goldLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.redox.2022.102570

publisherhttps://doi.org/10.1016/j.redox.2022.102570

publisherhttps://api.elsevier.com/content/article/PII:S2213231722003421?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2213231722003421?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36495698

Fields of Study

Ferroptosis and cancer prognosisGenomics, phytochemicals, and oxidative stressAutophagy in Disease and Therapy0301 basic medicine03 medical and health sciencesAnimalsMiceHumansNon-alcoholic Fatty Liver DiseaseNF-E2-Related Factor 2Kelch-Like ECH-Associated Protein 1AutophagosomesFatty AcidsObesityCell DeathDiet, High-FatMice, Inbred C57BLLiverLipid Metabolism

MeSH Terms

AutophagosomesKelch-Like ECH-Associated Protein 1AnimalsFatty AcidsHumansLiverMice, Inbred C57BLObesityCell DeathLipid MetabolismNF-E2-Related Factor 2MiceDiet, High-FatNon-alcoholic Fatty Liver Disease

Keywords

Fatty liverAutophagyDiseaseBiogenesisMetabolic syndromeAutophagosomeObesityProgrammed cell deathMedicineCancer researchEndocrinologyInternal medicineBiologyChemistryCell biologyBiochemistryGeneApoptosisFatty acidshigh fat dietNRF2MtorKeap1AmpkAKTAtg7NafldLiver SteatosisFerroptosisMedicine (General)QH301-705.5NF-E2-Related Factor 2Diet, High-FatMiceR5-920Non-alcoholic Fatty Liver DiseaseAnimalsHumansBiology (General)Kelch-Like ECH-Associated Protein 1Cell DeathAutophagosomesLipid MetabolismMice, Inbred C57BLLiverResearch Paper

Sustainable Development Goals

SDG 2: 2. Zero hungerSDG 3: 3. Good healthSDG 0: Good health and well-being

Additional file 1 of Nobiletin restores HFD-induced enteric nerve injury by regulating enteric glial activation and the GDNF/AKT/FOXO3a/P21 pathway

10.6084/m9.figshare.26757238.v1figshare

Additional file 1 of Nobiletin restores HFD-induced enteric nerve injury by regulating enteric glial activation and the GDNF/AKT/FOXO3a/P21 pathway

10.6084/m9.figshare.26757238figshare