Uremic toxin receptor NR1H3 contributes to hyperlipidemia- and chronic kidney disease-accelerated vascular inflammation, which is partially suppressed by novel YBX2 anti-ROS pathway is a research paper published in Redox Biology (2025). On theSindex it has a DataRank of 0.292. It has been cited 6 times.
Scored on demand from live citation data
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DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
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Base Score Contribution
0.292
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institute of Diabetes and Digestive and Kidney Diseases
Grant: DK113775
National Heart Lung and Blood Institute
Grant: HL163570-01A1
National Heart Lung and Blood Institute
Grant: 5T32HL091804-15
Integrative Cardiovascular Pathophysiology
National Heart Lung and Blood Institute
Grant: HL147565-01
NHLBI NIH HHS
Grant: T32 HL091804
NHLBI NIH HHS
Grant: R01 HL163570
NHLBI NIH HHS
Grant: R01 HL147565
National Institutes of Health
Grant: 1R01HL147565-01
IL-35 inhibits gut microbiota-produced uremic toxin-accelerated endothelial cell activation
National Institutes of Health
Grant: 5R01DK113775-04
CD40 monocyte in chronic kidney disease
National Institutes of Health
Grant: 1R01HL163570-01A1
LysoPI/GPR55 pathway promotes endothelial activation, vascular inflammation and atherosclerosis
National Institutes of Health
FWCI
2.52
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals