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The 1 –Cys peroxiredoxin, PRDX-6, suppresses an NHR-49-dependent pro-survival response, including the Flavin monooxygenase, FMO-2, that protects against fungal and bacterial infection

Redox Biology(2025)10.1016/j.redox.2025.103992Source: DataRank Database

The 1 –Cys peroxiredoxin, PRDX-6, suppresses an NHR-49-dependent pro-survival response, including the Flavin monooxygenase, FMO-2, that protects against fungal and bacterial infection is a research paper published in Redox Biology (2025). On theSindex it has a DataRank of 0.104. It has been cited 1 time.

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0.104DataRank · unranked

Scored on demand from live citation data

Open Access1 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (5 grants)Impact (FWCI 0.39)OA: goldTopics (46)IDs (PubMed, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

0.104

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

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Authors (10)

Emilia Dwyer,Jake B. Lewis,Madison S. Mortensen,Elise McDonald,Eloise Butler

Live enrichment

PubMed:41702017 OpenAlex:https://openalex.org/W7117472698

UK Research and Innovation Medical Research Council

Grant: G 0800082

Biotechnology and Biological Sciences Research Council

Grant: BB/T008695/1

NLD (Newcastle-Liverpool-Durham) Doctoral Training Partnership 3

Biotechnology and Biological Sciences Research Council

Grant: BB/T002484/1

Peroxiredoxinylation; a new post-translational modification promoting redox signal transduction?

National Institutes of Health

Grant: R 35GM152060

National Institutes of Health

Grant: P40 OD010440

FWCI

0.39

Citation Percentile

0.7%

Citation Trend

2026

goldLicense: cc-by

journalhttps://doi.org/10.1016/j.redox.2025.103992

publisherhttps://doi.org/10.1016/j.redox.2025.103992

publisherhttps://api.elsevier.com/content/article/PII:S2213231725005051?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2213231725005051?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/41702017

Fields of Study

Redox biology and oxidative stressGenetics, Aging, and Longevity in Model OrganismsSelenium in Biological Systems0301 basic medicine03 medical and health sciencesAnimalsCaenorhabditis elegansCaenorhabditis elegans ProteinsReactive Oxygen SpeciesOxygenasesPeroxiredoxinsOxidative StressMutationBacterial InfectionsReceptors, Cytoplasmic and Nuclear

MeSH Terms

AnimalsBacterial InfectionsMutationOxygenasesCaenorhabditis elegansReactive Oxygen SpeciesReceptors, Cytoplasmic and NuclearOxidative StressCaenorhabditis elegans ProteinsPeroxiredoxinsPeroxiredoxin VI

Keywords

Caenorhabditis elegansMutantReactive oxygen speciesPhenotypeStaphylococcus aureusGeneImmune systemFlavin groupOxidative stressResearch PaperAgingCandida albicansFerroptosisFlavin monooxygenaseFungal pathogenHNF4InfectionInnate immunityPPARαPeroxiredoxins