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Connections between endoplasmic reticulum stress-associated unfolded protein response, mitochondria, and autophagy in arsenic-induced carcinogenesis

Seminars in Cancer Biology(2021)10.1016/j.semcancer.2021.04.004Source: DataRank Database

Connections between endoplasmic reticulum stress-associated unfolded protein response, mitochondria, and autophagy in arsenic-induced carcinogenesis is a research paper published in Seminars in Cancer Biology (2021). On theSindex it has a DataRank of 0. It has been cited 125 times.

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Open Access125 citations

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Enrichment:Funding (10 grants)Impact (FWCI 9.33)OA: hybridTopics (39)Linked Datasets (4)DataCite Reuse (4)IDs (PubMed, PMC, OpenAlex)SDGs (1)

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Context only (not used in score)

Findable (2/2)

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Accessible (1/2)

Open Access

Interoperable (2/2)

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Reusable (0/3)

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Authors (2)

Fei Chen ORCID,Priya Wadgaonkar

Live enrichment

PubMed:33836253 PMC:PMC8492764 OpenAlex:https://openalex.org/W3141017649

National Institutes of Health

Grant: ES031822

National Institutes of Health

Grant: ES028335

National Institutes of Health

Grant: R01 ES028263

National Institutes of Health

Grant: P30 ES020957

NIEHS NIH HHS

Grant: R01 ES028335

NIEHS NIH HHS

Grant: R01 ES031822

National Institutes of Health

Grant: 5R01ES028263-04

Reduced reactive oxygen species and oxidative phosphorylation in arsenic-induced cancer stem cells

National Institutes of Health

Grant: 5R01ES031822-06

Dissecting Nrf2-dependent HIF1a activation mechanism in arsenic-induced cancer stem-like cells

National Institutes of Health

Grant: 5R01ES028335-02

Arsenic-induced miRNA-199 and miRNA-214 deplete mitochondrial DNA for the generation of cancer stem-like cells

National Institutes of Health

Grant: 5P30ES020957-07

Center for Urban Responses to Environmental Stressors (CURES)

FWCI

9.33

Citation Percentile

1.0%

Citation Trend

2021

2022

2023

2024

2025

2026

hybridLicense: cc-by-nc-nd

journalhttps://doi.org/10.1016/j.semcancer.2021.04.004

publisherhttps://doi.org/10.1016/j.semcancer.2021.04.004

publisherhttps://api.elsevier.com/content/article/PII:S1044579X2100105X?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S1044579X2100105X?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/33836253

Fields of Study

Endoplasmic Reticulum Stress and DiseaseAutophagy in Disease and TherapyArsenic contamination and mitigation0301 basic medicine0303 health sciences03 medical and health sciences

MeSH Terms

AnimalsArsenicAutophagyEnvironmental ExposureHumansMitochondriaUnfolded Protein ResponseEndoplasmic Reticulum StressCarcinogenesis

Keywords

Unfolded protein responseEndoplasmic reticulumAutophagyATF6CarcinogenesisBiologyCell biologyMitochondrionProteostasisEpigeneticsCancer researchCancerImmunologyApoptosisGeneticsArsenicER stressCancer Stem CellsUprAnimalsHumansEnvironmental ExposureEndoplasmic Reticulum StressMitochondria

Sustainable Development Goals

SDG 3: 3. Good health

Additional file 1 of Knockdown of NR3C1 inhibits the proliferation and migration of clear cell renal cell carcinoma through activating endoplasmic reticulum stress–mitophagy

10.6084/m9.figshare.26624509.v1figshare

Additional file 1 of Knockdown of NR3C1 inhibits the proliferation and migration of clear cell renal cell carcinoma through activating endoplasmic reticulum stress–mitophagy

10.6084/m9.figshare.26624509figshare

Additional file 2 of Knockdown of NR3C1 inhibits the proliferation and migration of clear cell renal cell carcinoma through activating endoplasmic reticulum stress–mitophagy

10.6084/m9.figshare.26624512.v1figshare

Additional file 2 of Knockdown of NR3C1 inhibits the proliferation and migration of clear cell renal cell carcinoma through activating endoplasmic reticulum stress–mitophagy

10.6084/m9.figshare.26624512figshare