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IL-8 and MCP-1/CCL2 regulate proteolytic activity in triple negative inflammatory breast cancer a mechanism that might be modulated by Src and Erk1/2

Toxicology and Applied Pharmacology(2020)10.1016/j.taap.2020.115092Source: DataRank Database

IL-8 and MCP-1/CCL2 regulate proteolytic activity in triple negative inflammatory breast cancer a mechanism that might be modulated by Src and Erk1/2 is a research paper published in Toxicology and Applied Pharmacology (2020). On theSindex it has a DataRank of 0. It has been cited 24 times.

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24 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 1.82)OA: closedTopics (49)IDs (PubMed, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (0/2)

Interoperable (0/2)

Reusable (0/3)

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Authors (8)

Eslam A. Elghonaimy ORCID,Mohamed El‐Shinawi ORCID,Mohamed Hosney,Martin Götte ORCID,Wendy A. Woodward ORCID

Live enrichment

PubMed:32512068 OpenAlex:https://openalex.org/W3033623035

National Institutes of Health

Grant: R21TW010952

National Institutes of Health

Grant: R03 TW008624

Horizon 2020 Framework Programme

Grant: 645756

Matrix glycans as multifunctional pathogenesis factors and therapeutic targets in cancer

FWCI

1.82

Citation Percentile

0.8%

Citation Trend

2021

2022

2023

2024

2025

2026

closedLicense: Elsevier TDM

publisherhttps://api.elsevier.com/content/article/PII:S0041008X20302167?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0041008X20302167?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.taap.2020.115092

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32512068

https://dx.doi.org/10.1016/j.taap.2020.115092

Fields of Study

Cell Adhesion Molecules ResearchChemokine receptors and signalingProtease and Inhibitor Mechanisms03 medical and health sciences0302 clinical medicine

MeSH Terms

DasatinibAdultAgedAntineoplastic AgentsFemaleHumansMiddle AgedInterleukin-8Genes, srcChemokine CCL2MAP Kinase Signaling SystemCell Line, TumorInflammatory Breast NeoplasmsTumor MicroenvironmentProteolysisTriple Negative Breast Neoplasms

Keywords

Inflammatory breast cancerCCL2Cancer researchChemokineProinflammatory cytokineTumor microenvironmentChemistryMedicineBreast cancerCancerImmunologyInflammationInternal medicineAdultMAP Kinase Signaling SystemInterleukin-8DasatinibAntineoplastic AgentsTriple Negative Breast NeoplasmsMiddle AgedGenes, srcCell Line, TumorProteolysisHumansFemaleInflammatory Breast NeoplasmsChemokine CCL2Aged

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

IL-8 and MCP-1/CCL2 regulate proteolytic activity in triple negative inflammatory breast cancer a mechanism that might be modulated by Src and Erk1/2

FAIR Checklist

Authors (8)

Funding (3 grants)

Impact

Open Access

Topics