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UPR, autophagy, and mitochondria crosstalk underlies the ER stress response

Trends in Biochemical Sciences(2015)10.1016/j.tibs.2015.01.002Source: DataRank Database

UPR, autophagy, and mitochondria crosstalk underlies the ER stress response is a research paper published in Trends in Biochemical Sciences (2015). On theSindex it has a DataRank of 1.1. It has been cited 1,134 times.

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1.1DataRank · unranked

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1,134 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 31.42)OA: bronzeTopics (38)Linked Datasets (25)DataCite Reuse (25)IDs (PubMed, PMC, OpenAlex)SDGs (1)

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Interoperable (2/2)

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Reusable (0/3)

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DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

1.1

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

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Authors (2)

Ze’ev A. Ronai,Daniela Senft

Live enrichment

PubMed:25656104 PMC:PMC4340752 OpenAlex:https://openalex.org/W2019263782

NCI NIH HHS

Grant: CA128814

NCI NIH HHS

Grant: P01 CA128814

NCI NIH HHS

Grant: P30 CA030199

FWCI

31.42

Citation Percentile

1.0%

Citation Trend

2015

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2026

bronzeLicense: https://www.elsevier.com/tdm/userlicense/1.0/

journalhttp://www.cell.com/article/S0968000415000031/pdf

publisherhttp://www.cell.com/article/S0968000415000031/pdf

publisherhttps://api.elsevier.com/content/article/PII:S0968000415000031?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0968000415000031?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.tibs.2015.01.002

Fields of Study

Endoplasmic Reticulum Stress and DiseaseAutophagy in Disease and TherapySirtuins and Resveratrol in MedicineAnimalsAutophagyCell Physiological PhenomenaEndoplasmic Reticulum StressHumansMitochondriaStress, PhysiologicalUnfolded Protein Response

MeSH Terms

AnimalsAutophagyCell Physiological PhenomenaHumansMitochondriaStress, PhysiologicalUnfolded Protein ResponseEndoplasmic Reticulum Stress

Keywords

AutophagyCrosstalkMitochondrionUnfolded protein responseCell biologyFight-or-flight responseBiologyCellular stress responseEndoplasmic reticulumApoptosisGeneticsEngineeringGeneElectronic engineeringMitochondriaHypoxiaUbiquitinER stressUpr

Sustainable Development Goals

SDG 0: Good health and well-being

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Additional file 1 of PERK/NRF2 and autophagy form a resistance mechanism against G9a inhibition in leukemia stem cells

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Additional file 1 of Inhibition of ERAD synergizes with FTS to eradicate pancreatic cancer cells

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Additional file 1 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

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Additional file 1 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618840figshare

Additional file 2 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618843.v1figshare

Additional file 2 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618843figshare

Additional file 3 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618846.v1figshare

Additional file 4 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618849.v1figshare

Additional file 3 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618846figshare

Additional file 4 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618849figshare

Additional file 5 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618852.v1figshare

Additional file 5 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618852figshare

Additional file 6 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618855.v1figshare

Additional file 6 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618855figshare

Additional file 7 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618858.v1figshare

Additional file 7 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618858figshare

Additional file 8 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618861.v1figshare

Additional file 8 of rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

10.6084/m9.figshare.22618861figshare

UPR, autophagy, and mitochondria crosstalk underlies the ER stress response

FAIR Checklist

DataRank Breakdown

Authors (2)

Funding (3 grants)

Impact

Open Access

Topics

Linked Datasets (25)