Non-enzymatic glycoxidation linked with nutrition enhances the tumorigenic capacity of prostate cancer epithelia through AGE mediated activation of RAGE in cancer associated fibroblasts is a research paper published in Translational Oncology (2022). On theSindex it has a DataRank of 0. It has been cited 26 times.
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Medical University of South Carolina
Grant: P30 CA138313
National Cancer Institute
Grant: R01CA245143
National Cancer Institute
Grant: R21CA176135
National Cancer Institute
Grant: R21CA194469
National Cancer Institute
Grant: R21CA258139
National Cancer Institute
Grant: U54CA210962
NCCIH NIH HHS
Grant: F31 AT011158
NCI NIH HHS
Grant: U54 CA210963
National Institutes of Health
Grant: 7R01CA245143-03
Lifestyle associated reactive metabolites and their negative impact on breast cancer risk
National Institutes of Health
Grant: 1R21CA258139-01A1
The effects of time restricted feeding on AGE-RAGE signaling in women at high risk for breast cancer
National Institutes of Health
Grant: 5P30CA138313-09
Medical University of South Carolina - Cancer Center Support Grant
National Institutes of Health
Grant: 3U54CA210962-05S1
South Carolina Cancer Disparities Research Center (SC CADRE)
National Institutes of Health
Grant: 1R21CA194469-01
(PQ3) AGEs and Race Specific Tumor Immune Response in Prostate Cancer
National Institutes of Health
Grant: 1F31AT011158-01
Complimentary NMR and Bio-Assay Guided Screening of Edible Marine Algae for AGE Inhibitors to Control Glycative Stress and Reduce Risk of Neurodegeneration.
National Institutes of Health
Grant: 1R21CA176135-01
Glycation as a Mechanism Promoting Cancer Disparity
National Institutes of Health
FWCI
2.60
Citation Percentile
0.9%
Citation Trend
Fields of Study
Keywords
Sustainable Development Goals
Additional file 3 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 4 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 4 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 1 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 2 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 1 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 3 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland
Additional file 2 of Pubertal exposure to dietary advanced glycation end products disrupts ductal morphogenesis and induces atypical hyperplasia in the mammary gland