Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

TDP-43 proteinopathy in ALS is triggered by loss of ASRGL1 and associated with HML-2 expression

Nature Communications(2024)10.1038/s41467-024-48488-7Source: DataRank Database

TDP-43 proteinopathy in ALS is triggered by loss of ASRGL1 and associated with HML-2 expression is a research paper published in Nature Communications (2024). On theSindex it has a DataRank of 0. It has been cited 29 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access29 citations

Download PDF

Cite:

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (2 grants)Impact (FWCI 7.69)OA: goldTopics (45)IDs (PubMed, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (27)

Saeed Fathi ORCID,Cyrus Rastegar ORCID,Elena Rita Simula ORCID,Tara T. Doucet-O’Hare ORCID,Yong-Han Hank Cheng ORCID

Live enrichment

PubMed:38755145 OpenAlex:https://openalex.org/W4396950248

U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke

Grant: NS003130

BLRD VA

Grant: I01 BX002466

FWCI

7.69

Citation Percentile

1.0%

Citation Trend

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1038/s41467-024-48488-7

publisherhttps://doi.org/10.1038/s41467-024-48488-7

publisherhttps://www.nature.com/articles/s41467-024-48488-7.pdf

publisherhttps://www.nature.com/articles/s41467-024-48488-7

repositoryhttps://pubmed.ncbi.nlm.nih.gov/38755145

Fields of Study

Amyotrophic Lateral Sclerosis ResearchNeurogenetic and Muscular Disorders ResearchParkinson's Disease Mechanisms and TreatmentsAnimalsFemaleHumansMaleMiceAmyotrophic Lateral SclerosisAsparaginaseAutoantigensBrainDNA-Binding ProteinsMotor CortexNeuronsTDP-43 ProteinopathiesEndogenous Retroviruses

MeSH Terms

Amyotrophic Lateral SclerosisAnimalsAsparaginaseAutoantigensBrainDNA-Binding ProteinsFemaleHumansMaleMotor CortexNeuronsEndogenous RetrovirusesMiceTDP-43 Proteinopathies

Keywords

Gene silencingBiologyCell biologyCytoplasmAmyotrophic lateral sclerosisEndogenous retrovirusNeurodegenerationSmall hairpin RNAGene knockdownMolecular biologyGeneGeneticsPathologyMedicine