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CTLA-4-expressing ILC3s restrain interleukin-23-mediated inflammation

Nature(2024)10.1038/s41586-024-07537-3Source: DataRank Database

CTLA-4-expressing ILC3s restrain interleukin-23-mediated inflammation is a research paper published in Nature (2024). On theSindex it has a DataRank of 0.938. It has been cited 44 times, with 40 citing works in its 1-hop citation network.

N/A

0.938DataRank · unranked

Scored on demand from live citation data

Deposits data Open Access44 citations · 40 citing works in its network

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Data availability

Repositories this paper deposited data in (declared in PubMed).

GEOGSE229976

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (19 grants)Impact (FWCI 6.93)OA: greenTopics (79)IDs (PubMed, PMC, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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DataRank Breakdown

Base Score 61%Citation Network 39%

Base Score Contribution

0.571

From this paper's citation signal

Citation Network Contribution

0.367

From 21 citing papers with measurable signal

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Top 5 citers driving the network score

Ranked by each citer's contribution to N(p) — log1p(C_q) divided by its reference count — out of 40 citers.

Highlights of 2024: The multifaceted roles of ILC3s in mucosal immunity and inflammation
Immunology and Cell Biology20251 citations10 referencesContributes 0.069
Epidemiology, pathogenesis, diagnosis, and treatment of inflammatory bowel disease: Insights from the past two years
Chinese Medical Journal202565 citations117 referencesContributes 0.036
Macrophages regulate PD-1 and CTLA-4 expression on ILC2s and their responsiveness in the tumor microenvironment
Cellular and Molecular Immunology20255 citations51 referencesContributes 0.035
PD1+ innate lymphoid cells 3 predict JAK-dependent inflammation in rheumatoid arthritis
Journal of Autoimmunity20254 citations50 referencesContributes 0.032
Expansion of Interleukin‐22‐Producing Type 3 Innate Lymphoid Cells in the Gut of Tristetraprolin‐Deficient Mice
European Journal of Immunology20253 citations45 referencesContributes 0.031

Authors (32)

Ann Mary Joseph ORCID,Jordan Zhou ORCID,Veronika Horn ORCID,Jazib Uddin ORCID,Mengze Lyu ORCID

Live enrichment

PubMed:38867048 PMC:PMC11298788 OpenAlex:https://openalex.org/W4399567403

NIAID NIH HHS

Grant: R01 AI123368

NIAID NIH HHS

Grant: R01 AI143842

NIAID NIH HHS

Grant: R01 AI162936

NIAID NIH HHS

Grant: R37 AI174468

NIAID NIH HHS

Grant: U01 AI095608

NIDDK NIH HHS

Grant: F32 DK136248

NCI NIH HHS

Grant: R01 CA274534

NIDDK NIH HHS

Grant: T32 DK116970

NIAID NIH HHS

Grant: R01 AI145989

National Institutes of Health

Grant: 1R37AI174468-01A1

Novel mechanisms protecting the gut from TNF

National Institutes of Health

Grant: 5R01AI145989-02

Interleukin-2 regulation of mucosal inflammation

National Institutes of Health

Grant: 1F32DK136248-01

Regulation of Enteric Nervous System Function by Group 3 Innate Lymphoid Cells

National Institutes of Health

Grant: 1R01AI162936-01

Innate immune regulation of neuroinflammation

National Institutes of Health

Grant: 1R01AI143842-01

Defining a novel mechanism of mucosal healing

National Institutes of Health

Grant: 5T32DK116970-03

Multidisciplinary Research Training in Gastroenterology and Hepatology

National Institutes of Health

Grant: 5U01AI095608-14

Regulation of mucosal immunity by neuronal pathways

National Institutes of Health

Grant: 3R01CA274534-03S1

Innate lymphoid cell regulation of the host-microbiota interactions in cancer

National Institutes of Health

Grant: 2R01AI123368-06

Immune regulation of intestinal health and disease

Wellcome Trust

Grant: unidentified

unidentified

FWCI

6.93

Citation Percentile

1.0%

Citation Trend

2024

2025

2026

greenLicense: Springer Nature TDM

repositoryhttps://amu.hal.science/hal-04934644/document

publisherhttps://www.nature.com/articles/s41586-024-07537-3.pdf

publisherhttps://www.nature.com/articles/s41586-024-07537-3

repositoryhttps://amu.hal.science/hal-04934644

Fields of Study

IL-33, ST2, and ILC PathwaysWhipple's Disease and InterleukinsImmune Cell Function and Interaction03 medical and health sciences0302 clinical medicineAnimalsFemaleHumansMaleMiceCTLA-4 AntigenForkhead Box Protein O1Gastrointestinal MicrobiomeImmunity, InnateInflammationInterleukin-23IntestinesLymphocytesMice, Inbred C57BLMyeloid CellsSingle-Cell Gene Expression AnalysisSTAT3 Transcription FactorT-Lymphocytes, RegulatoryB7-H1 AntigenInterleukin-23 Subunit p19

MeSH Terms

Gastrointestinal MicrobiomeForkhead Box Protein O1RNA-SeqSingle-Cell Gene Expression AnalysisAnimalsFemaleHumansImmunity, InnateInflammationIntestinesLymphocytesMaleMice, Inbred C57BLMyeloid CellsT-Lymphocytes, RegulatorySTAT3 Transcription FactorMiceInterleukin-23Interleukin-23 Subunit p19Single-Cell AnalysisB7-H1 AntigenCTLA-4 Antigen

Keywords

ImmunologyInflammationInnate lymphoid cellDownregulation and upregulationBiologyCTLA-4Immune systemCancer researchT cellImmunityMaleSTAT3 Transcription FactorInterleukin-23T-Lymphocytes, RegulatoryB7-H1 AntigenMiceAnimalsHumansCTLA-4 AntigenMyeloid CellsLymphocytesForkhead Box Protein O1Immunity, InnateSingle-Cell Gene Expression AnalysisGastrointestinal Microbiome[SDV] Life Sciences [q-bio]IntestinesMice, Inbred C57BLInterleukin-23 Subunit p19CytokinesMucosal immunologyFemale

Sustainable Development Goals

SDG 0: Zero hungerSDG 0: Good health and well-being