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The phosphatidylinositol-transfer protein Nir3 promotes PI(4,5)P2 replenishment in response to TCR signaling during T cell development and survival

Nature Immunology(2022)10.1038/s41590-022-01372-2Source: DataRank Database

The phosphatidylinositol-transfer protein Nir3 promotes PI(4,5)P2 replenishment in response to TCR signaling during T cell development and survival is a research paper published in Nature Immunology (2022). On theSindex it has a DataRank of 0. It has been cited 19 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access19 citations

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Linked data & code

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (5 grants)OA: hybridTopics (7)IDs (PubMed, PMC)Annotations (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (6)

Ynes Helou,Krishna Shrinivas ORCID,Jen Liou ORCID,Byron B. Au‐Yeung ORCID,Arthur Weiss ORCID

Live enrichment

PubMed:36581712 PMC:PMC9810531

U.S. Department of Health & Human Services | NIH | National Institute of Allergy and Infectious Diseases

Grant: R37 AI114575

U.S. Department of Health & Human Services | NIH | National Institute of General Medical Sciences

Grant: R01 GM 144479

U.S. Department of Health & Human Services | NIH | National Institute of Arthritis and Musculoskeletal and Skin Diseases

Grant: K01 AR065481

NIDDK NIH HHS

Grant: P30 DK063720

Howard Hughes Medical Institute

hybridLicense: cc-by

publisherhttps://www.nature.com/articles/s41590-022-01372-2.pdf

publisherhttps://www.nature.com/articles/s41590-022-01372-2

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/9810531

Europe_PMChttps://europepmc.org/articles/PMC9810531

Europe_PMChttps://europepmc.org/articles/PMC9810531?pdf=render

MeSH Terms

Cell MembraneAnimalsMicePhosphatidylinositolsPhospholipid Transfer ProteinsReceptors, Antigen, T-CellSignal Transduction