Articular cartilage regeneration by activated skeletal stem cells is a research paper published in Nature Medicine (2020). On theSindex it has a DataRank of 0.885. It has been cited 364 times.
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Base Score Contribution
0.885
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →U.S. Department of Health & Human Services | NIH | National Institute on Aging
Grant: R00AG049958
NIGMS NIH HHS
Grant: R01 GM123069
NIDCR NIH HHS
Grant: R21 DE019274
NIDCR NIH HHS
Grant: U24 DE026914
NIA NIH HHS
Grant: K99 AG049958
NIDCR NIH HHS
Grant: R21 DE024230
NIGMS NIH HHS
Grant: T32 GM119995
National Institute for Health Research (NIHR)
Grant: ACF-2019-06-008
NIDCR NIH HHS
Grant: R01 DE021683
NIDCR NIH HHS
Grant: R56 DE025597
NHLBI NIH HHS
Grant: U01 HL099776
NIAMS NIH HHS
Grant: R01 AR071379
NIDCR NIH HHS
Grant: R01 DE026730
NIDCR NIH HHS
Grant: R01 DE027323
NIH HHS
Grant: S10 OD021514
National Institutes of Health
Grant: 5R01DE026730-02
Cellular and Mechanical Mechanisms Regulating Mandibular Distraction Osteogenesis
National Institutes of Health
Grant: 5R01DE027323-05
Identifying the human skeletal stem cell.
National Institutes of Health
Grant: 1S10OD021514-01
Bio-AFM for combined light and atomic force imaging
National Institutes of Health
Grant: 7R01GM123069-04
Developing New Diagnostic and Timed, TAK1 Specific Treatment Strategies for Trauma Induced Heterotopic Ossification
National Institutes of Health
Grant: 5R01AR071379-04
Targeting Molecular and Cellular Mediators of Inflammation to Prevent Pathologic Cell Differentiation and Heterotopic Ossification
National Institutes of Health
Grant: 1R21DE024230-01
Enhancing Bcl-2 Expression for Bone Regeneration.
National Institutes of Health
Grant: 1K99AG049958-01A1
Mechanisms of Skeletal Stem Cell Aging
National Institutes of Health
Grant: 5R00AG049958-04
Mechanisms of Skeletal Stem Cell Aging
National Institutes of Health
Grant: 4R01DE021683-05
Osteogenic Enrichment of Adipose Derived Stromal Cells
National Institutes of Health
Grant: 5U24DE026914-03
Center for Dental, Oral, and Craniofacial Tissue and Organ Regeneration (C-DOCTOR)
National Institutes of Health
Grant: 1R56DE025597-01A1
Identification and Specification of the Human Skeletal Stem Cell
National Institutes of Health
Grant: 5R21DE019274-02
Enhanced Calvarial Regeneration Via RNAi-Mediated Suppression of BMP Antagonism
National Institutes of Health
Grant: 5U01HL099776-06
Induced pluripotent stem cells in the understanding and treatment of heart diseas
Arthritis National Research Foundation
American Federation for Aging Research
Howard Hughes Medical Institute
FWCI
35.03
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 1 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 4 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 4 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 5 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 5 of Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Additional file 1 of Silencing Smad7 potentiates BMP2-induced chondrogenic differentiation and inhibits endochondral ossification in human synovial-derived mesenchymal stromal cells
Additional file 1 of Silencing Smad7 potentiates BMP2-induced chondrogenic differentiation and inhibits endochondral ossification in human synovial-derived mesenchymal stromal cells
Additional file 1 of Intrauterine infusion of clinically graded human umbilical cord-derived mesenchymal stem cells for the treatment of poor healing after uterine injury: a phase I clinical trial
Additional file 1 of Intrauterine infusion of clinically graded human umbilical cord-derived mesenchymal stem cells for the treatment of poor healing after uterine injury: a phase I clinical trial
Additional file 2 of Intrauterine infusion of clinically graded human umbilical cord-derived mesenchymal stem cells for the treatment of poor healing after uterine injury: a phase I clinical trial
Additional file 2 of Intrauterine infusion of clinically graded human umbilical cord-derived mesenchymal stem cells for the treatment of poor healing after uterine injury: a phase I clinical trial
Additional file 1 of Hierarchical porous ECM scaffolds incorporating GDF-5 fabricated by cryogenic 3D printing to promote articular cartilage regeneration
Additional file 1 of Hierarchical porous ECM scaffolds incorporating GDF-5 fabricated by cryogenic 3D printing to promote articular cartilage regeneration
Additional file 1 of Emilin2 marks the target region for mesenchymal cell accumulation in bone regeneration
Additional file 1 of Emilin2 marks the target region for mesenchymal cell accumulation in bone regeneration
Additional file 2 of Emilin2 marks the target region for mesenchymal cell accumulation in bone regeneration
Additional file 2 of Emilin2 marks the target region for mesenchymal cell accumulation in bone regeneration
Additional file 4 of Identification of therapeutic targets in osteoarthritis by combining heterogeneous transcriptional datasets, drug-induced expression profiles, and known drug-target interactions
Additional file 4 of Identification of therapeutic targets in osteoarthritis by combining heterogeneous transcriptional datasets, drug-induced expression profiles, and known drug-target interactions