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Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathway

Scientific Reports(2021)10.1038/s41598-021-00282-xSource: DataRank Database

Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathway is a research paper published in Scientific Reports (2021). On theSindex it has a DataRank of 0. It has been cited 2 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access2 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (6 grants)Impact (FWCI 0.07)OA: goldTopics (63)IDs (PubMed, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (7)

Montamas Suntravat ORCID,Elda E. Sánchez ORCID,Beata M. Wolska ORCID,R. John Solaro,Pieter P. de Tombe ORCID

Live enrichment

PubMed:34707114 OpenAlex:https://openalex.org/W3209729759

NIH/ORIP Viper Resource

Grant: 5P40OD010960-14

RO1; University of Illinois at Chicago, USA

Grant: 128468

PO1

Grant: HL062426

National Institutes of Health

Grant: 5P40OD010960-17

Viper Resource Grant at Texas A&M University-Kingsville

National Institutes of Health

Grant: 5P01HL062426-05

INTEGRATED MECHANISMS OF CARDIAC MALADAPTATION

National Institutes of Health

Grant: 2R15HL137134-02

The acute effects of snake venom CRiSP toxins on blood and lymphatic endothelial cell permeability: new insights into the pathophysiology of snakebite

FWCI

0.07

Citation Percentile

0.4%

Citation Trend

2024

2026

goldLicense: cc-by

journalhttps://www.nature.com/articles/s41598-021-00282-x.pdf

publisherhttps://www.nature.com/articles/s41598-021-00282-x.pdf

publisherhttps://www.nature.com/articles/s41598-021-00282-x

journalhttps://doi.org/10.1038/s41598-021-00282-x

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34707114

Fields of Study

Ion channel regulation and functionCardiac electrophysiology and arrhythmiasCardiovascular Effects of Exercise0301 basic medicine03 medical and health sciences0303 health sciences

MeSH Terms

Adenosine TriphosphatasesAdrenergic beta-AntagonistsAnimalsCalciumCells, CulturedCobra Cardiotoxin ProteinsMaleMyofibrilsPhosphorylationIon TransportRats, Sprague-DawleyCyclic AMP-Dependent Protein KinasesCalcium SignalingMyocytes, CardiacRats

Keywords

CardiotoxinMyofilamentMyofibrilInternal medicineInotropeMyocyteEndocrinologyChemistryPhosphorylationPharmacologyCell biologyBiologyMedicineSkeletal muscleMaleScienceAdrenergic beta-AntagonistsCobra Cardiotoxin ProteinsArticleRats, Sprague-DawleyMyofibrilsBiochemistry, Genetics and Molecular BiologyHealth SciencesAnimalsMyocytes, CardiacCalcium SignalingMolecular BiologyCells, CulturedAdenosine TriphosphatasesIon TransportDiagnosis and Management of Arrhythmogenic Right Ventricular CardiomyopathyQRLife SciencesMolecular Mechanisms of Cardiac ArrhythmiasCyclic AMP-Dependent Protein KinasesRats[SDV] Life Sciences [q-bio]Molecular Mechanisms of Ion Channels RegulationArrhythmogenic Right Ventricular CardiomyopathyCalciumCardiology and Cardiovascular Medicine

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being