Rad52 inactivation is synthetically lethal with BRCA2 deficiency is a research paper published in Proceedings of the National Academy of Sciences (2011). On theSindex it has a DataRank of 9.9. It has been cited 341 times, with 200 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Base Score Contribution
0.875
From this paper's citation signal
Citation Network Contribution
9.0
From 200 citing papers with measurable signal
NCI NIH HHS
Grant: CA107640
NCI NIH HHS
Grant: R01 CA123232
NCI NIH HHS
Grant: CA123232
NCI NIH HHS
Grant: R01 CA107640
FWCI
6.35
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway
Additional file 1 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway
Additional file 2 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway
Additional file 2 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway
Additional file 3 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway
Additional file 3 of Nickel nanoparticle-induced cell transformation: involvement of DNA damage and DNA repair defect through HIF-1α/miR-210/Rad52 pathway