Macrophage metabolic reprogramming presents a therapeutic target in lupus nephritis is a research paper published in Proceedings of the National Academy of Sciences (2020). On theSindex it has a DataRank of 0. It has been cited 221 times.
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China Scholarship Council
Grant: 201408060241
RCUK | Medical Research Council
Grant: MR/N024907/1
Arthritis Research UK
Grant: 21777
Wellcome
Grant: 106809/Z/15/Z
NIHR Blood and Transplant Research Unit
Grant: NIHR BTRU-2014-10027
Kidney Research UK
Grant: TF_013_20171124
Medical Research Council
Grant: MC_UU_00015/3
Wellcome Trust
Grant: 110158/Z/15/Z
National Institute for Health Research (NIHR)
Grant: CL-2015-14-001
Intramural NIH HHS
Grant: ZIA AR041199
Medical Research Council
Grant: MR/S035842/1
FWCI
14.93
Citation Percentile
1.0%
Influential Citations
8
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Integrative analysis of single-cell and bulk transcriptome data reveal the significant role of macrophages in lupus nephritis
Additional file 1 of Integrative analysis of single-cell and bulk transcriptome data reveal the significant role of macrophages in lupus nephritis
Additional file 1 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 1 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 2 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 2 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 3 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 3 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 4 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 4 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 1 of Excessive IL-15 promotes cytotoxic CD4 + CD28− T cell-mediated renal injury in lupus nephritis
Additional file 1 of Excessive IL-15 promotes cytotoxic CD4 + CD28− T cell-mediated renal injury in lupus nephritis
Additional file 1 of Macrophage subpopulations in pediatric patients with lupus nephritis and other inflammatory diseases affecting the kidney
Additional file 1 of Macrophage subpopulations in pediatric patients with lupus nephritis and other inflammatory diseases affecting the kidney
Additional file 2 of Macrophage subpopulations in pediatric patients with lupus nephritis and other inflammatory diseases affecting the kidney
Additional file 2 of Macrophage subpopulations in pediatric patients with lupus nephritis and other inflammatory diseases affecting the kidney
Additional file 2 of Causal effect of gut microbiota and diabetic nephropathy: a Mendelian randomization study
Additional file 2 of Causal effect of gut microbiota and diabetic nephropathy: a Mendelian randomization study
Additional file 4 of Investigation of ferroptosis-associated molecular subtypes and immunological characteristics in lupus nephritis based on artificial neural network learning
Additional file 3 of Investigation of ferroptosis-associated molecular subtypes and immunological characteristics in lupus nephritis based on artificial neural network learning