Identification of a brainstem locus that inhibits tumor necrosis factor is a research paper published in Proceedings of the National Academy of Sciences (2020). On theSindex it has a DataRank of 3.1. It has been cited 133 times, with 98 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Base Score Contribution
0.735
From this paper's citation signal
Citation Network Contribution
2.4
From 83 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 98 citers.
NIH/NIGMS
Grant: 1R01GM132672-01
The ion channel TRPA1 is required for suppression of inflammation in sepsis
NIH/NIGMS
Grant: 1R35GM118182-01
Molecular Basis of Bioelectronic Medicine
NIH/NIGMS
Grant: 1R01GM128008-01
Brainstem Cholinergic Circuitries in the Inflammatory Reflex during Sepsis
NIH/NIAID
Grant: 1P01AI102852-01A1
Administrative Core
NIAID NIH HHS
Grant: P01 AI102852
NIGMS NIH HHS
Grant: R01 GM132672
NIGMS NIH HHS
Grant: R35 GM118182
NIGMS NIH HHS
Grant: R01 GM128008
FWCI
6.25
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 2 of M1 cholinergic signaling in the brain modulates cytokine levels and splenic cell sub-phenotypes following cecal ligation and puncture
Additional file 2 of M1 cholinergic signaling in the brain modulates cytokine levels and splenic cell sub-phenotypes following cecal ligation and puncture
Additional file 1 of M1 cholinergic signaling in the brain modulates cytokine levels and splenic cell sub-phenotypes following cecal ligation and puncture
Additional file 1 of M1 cholinergic signaling in the brain modulates cytokine levels and splenic cell sub-phenotypes following cecal ligation and puncture