Targeting the coronavirus nucleocapsid protein through GSK-3 inhibition is a research paper published in Proceedings of the National Academy of Sciences (2021). On theSindex it has a DataRank of 2.7. It has been cited 102 times, with 88 citing works in its 1-hop citation network.
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Base Score Contribution
0.695
From this paper's citation signal
Citation Network Contribution
2.0
From 76 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 88 citers.
Office of Extramural Research, National Institutes of Health
Grant: R21AI161678
Office of Extramural Research, National Institutes of Health
Grant: 1R01HL141759
Office of Extramural Research, National Institutes of Health
Grant: 1R01GM115517
UCLA DGSOM and Broad Stem Cell Research Center Institutional Award
Grant: OCRC #20-15
Office of Extramural Research, National Institutes of Health
Grant: 1R01EY032149
California Institute of Regenerative Medicine
Grant: TRAN1COVID19-11975
Office of Extramural Research, National Institutes of Health
Grant: 5R01AI140539
Office of Extramural Research, National Institutes of Health
Grant: 1R01AI1502461
Office of Extramural Research, National Institutes of Health
Grant: R01AI152362
Dean's Innovation Fund
Grant: N/A
Burroughs Wellcome Fund
Grant: Pathogenesis of Infectious Diseases
NIGMS NIH HHS
Grant: R01 GM115517
NIDDK NIH HHS
Grant: R01 DK132735
NIDDK NIH HHS
Grant: T32 DK007780
NIAID NIH HHS
Grant: R01 AI140539
NHLBI NIH HHS
Grant: R01 HL141759
NEI NIH HHS
Grant: R01 EY032149
National Institutes of Health
Grant: 5R01GM115517-02
An unexpected signaling output for the tumor suppressor APC
National Institutes of Health
Grant: 5R01EY032149-03
Interplay between AMPK and Hippo Signaling Regulates Ocular Antiviral Response to Zika virus infection
National Institutes of Health
Grant: 1R01AI140539-01
Defining the functional interface between the ER and flaviviruses
National Institutes of Health
Grant: 5R01HL141759-03
Maintenance and expansion of long-term hematopoietic stem cells
National Institutes of Health
Grant: 1R01AI152362-01
Defining the role of microbiota-derived cyclic dinucleotides in priming antiviral immune defenses.
National Institutes of Health
Grant: 1R21AI161678-01
Targeting Coronavirus through Nucleocapsid Phosphorylation
Wellcome Trust
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 1 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 2 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 2 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 3 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 3 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 4 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 4 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 5 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 5 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 6 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway
Additional file 6 of SARS-CoV-2 causes dysfunction in human iPSC-derived brain microvascular endothelial cells potentially by modulating the Wnt signaling pathway